Genetic Restoration of Heme Oxygenase-1 Expression Protects from Type 1 Diabetes in NOD Mice
出版年份 2019 全文链接
标题
Genetic Restoration of Heme Oxygenase-1 Expression Protects from Type 1 Diabetes in NOD Mice
作者
关键词
-
出版物
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES
Volume 20, Issue 7, Pages 1676
出版商
MDPI AG
发表日期
2019-04-04
DOI
10.3390/ijms20071676
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Upregulation of Heme Oxygenase-1 Endues Immature Dendritic Cells With More Potent and Durable Immunoregulatory Properties and Promotes Engraftment in a Stringent Mouse Cardiac Allotransplant Model
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- Heme Oxygenase-1-Expressing Dendritic Cells Promote Foxp3+ Regulatory T Cell Differentiation and Induce Less Severe Airway Inflammation in Murine Models
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- Carbon monoxide decreases endosome-lysosome fusion and inhibits soluble antigen presentation by dendritic cells to T cells.
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- Tolerogenic dendritic cells actively inhibit T cells through heme oxygenase-1 in rodents and in nonhuman primates
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- Carbon Monoxide Inhibits TLR-Induced Dendritic Cell Immunogenicity
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- Pivotal Advance: Heme oxygenase 1 expression by human CD4+ T cells is not sufficient for their development of immunoregulatory capacity
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- Genome-wide association study and meta-analysis find that over 40 loci affect risk of type 1 diabetes
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- Suppression by CD4+CD25+ Regulatory T Cells Is Dependent on Expression of Heme Oxygenase-1 in Antigen-Presenting Cells
- (2008) James F. George et al. AMERICAN JOURNAL OF PATHOLOGY
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