Lamin A buffers CK2 kinase activity to modulate aging in a progeria mouse model.

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A Werner syndrome stem cell model unveils heterochromatin alterations as a driver of human aging

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HP1α mediates defective heterochromatin repair and accelerates senescence inZmpste24-deficient cells

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Lamin A/C and emerin regulate MKL1–SRF activity by modulating actin dynamics

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Structural and functional insights into the regulation mechanism of CK2 by IP6 and the intrinsically disordered protein Nopp140

(2013) W.-K. Lee et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Targeting Isoprenylcysteine Methylation Ameliorates Disease in a Mouse Model of Progeria

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Prelamin A causes progeria through cell-extrinsic mechanisms and prevents cancer invasion

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Role of polyamines in determining the cellular response to chemotherapeutic agents: modulation of protein kinase CK2 expression and activity

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First Inactive Conformation of CK2α, the Catalytic Subunit of Protein Kinase CK2

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Induction of autophagy by spermidine promotes longevity

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Nuclear lamins: major factors in the structural organization and function of the nucleus and chromatin

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Premature aging in mice activates a systemic metabolic response involving autophagy induction

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Evaluating CK2 activity with the antibody specific for the CK2-phosphorylated form of a kinase-targeting cochaperone Cdc37

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HP1-β mobilization promotes chromatin changes that initiate the DNA damage response

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Tethering by lamin A stabilizes and targets the ING1 tumour suppressor

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