Long noncoding RNA UCA1 inhibits ischaemia/reperfusion injury induced cardiomyocytes apoptosis via suppression of endoplasmic reticulum stress

Long Noncoding RNAs and MicroRNAs in Cardiovascular Pathophysiology

(2015) T. Thum et al.   CIRCULATION RESEARCH

Pharmacologic Inhibition of the NLRP3 Inflammasome Preserves Cardiac Function After Ischemic and Nonischemic Injury in the Mouse

(2015) Carlo Marchetti et al.   JOURNAL OF CARDIOVASCULAR PHARMACOLOGY

Long noncoding RNAs in cardiac development and ageing

(2015)   Nature Reviews Cardiology

Breast carcinoma arising from sclerosing adenosis: a clinicopathologic and immunohistochemical analysis of 106 cases

(2014) Bao-Hua Yu et al.   PATHOLOGY

Mfn2 modulates the UPR and mitochondrial function via repression of PERK

(2013) Juan Pablo Muñoz et al.   EMBO JOURNAL

Long non-coding RNA UCA1 regulated cell cycle distribution via CREB through PI3-K dependent pathway in bladder carcinoma cells

(2012) Chen Yang et al.   GENE

β-Adrenergic receptor stimulation induces endoplasmic reticulum stress in adult cardiac myocytes: role in apoptosis

(2012) Suman Dalal et al.   MOLECULAR AND CELLULAR BIOCHEMISTRY

SDF-1/CXCR4 mediates acute protection of cardiac function through myocardial STAT3 signaling following global ischemia/reperfusion injury

(2011) Chunyan Huang et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY

The role of oxidized cytochromecin regulating mitochondrial reactive oxygen species production and its perturbation in ischaemia

(2011) Philippe Pasdois et al.   BIOCHEMICAL JOURNAL

Endoplasmic reticulum stress response in an INS-1 pancreatic β-cell line with inducible expression of a folding-deficient proinsulin

(2010) Taila Hartley et al.   BMC CELL BIOLOGY

Short-term dietary restriction and fasting precondition against ischemia reperfusion injury in mice

(2009) James R. Mitchell et al.   AGING CELL

Metallothionein alleviates oxidative stress-induced endoplasmic reticulum stress and myocardial dysfunction

(2009) Rui Guo et al.   JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY

Inhibition of Mitochondrial Permeability Transition Pore Opening by Ischemic Preconditioning Is Probably Mediated by Reduction of Oxidative Stress Rather Than Mitochondrial Protein Phosphorylation

(2008) Samantha J. Clarke et al.   CIRCULATION RESEARCH