Effect of Long-Term Exercise Training on lncRNAs Expression in the Vascular Injury of Insulin Resistance
出版年份 2018 全文链接
标题
Effect of Long-Term Exercise Training on lncRNAs Expression in the Vascular Injury of Insulin Resistance
作者
关键词
lncRNA, Insulin resistance, Vascular injury, Exercise
出版物
Journal of Cardiovascular Translational Research
Volume 11, Issue 6, Pages 459-469
出版商
Springer Nature
发表日期
2018-10-09
DOI
10.1007/s12265-018-9830-0
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Atherosclerotic Cardiovascular Disease in South Asians in the United States: Epidemiology, Risk Factors, and Treatments: A Scientific Statement From the American Heart Association
- (2018) Annabelle Santos Volgman et al. CIRCULATION
- Construction of competitive endogenous RNA network reveals regulatory role of long non-coding RNAs in type 2 diabetes mellitus
- (2017) Zijing Lin et al. JOURNAL OF CELLULAR AND MOLECULAR MEDICINE
- Protein quantitative trait locus study in obesity during weight-loss identifies a leptin regulator
- (2017) Jérôme Carayol et al. Nature Communications
- Insulin Resistance and Endothelial Dysfunction Constitute a Common Therapeutic Target in Cardiometabolic Disorders
- (2016) A. Janus et al. MEDIATORS OF INFLAMMATION
- Impact of high fat diet on long non-coding RNAs and messenger RNAs expression in the aortas of ApoE(−/−) mice
- (2016) Mei-hua Bao et al. Scientific Reports
- Insulin resistance: an additional risk factor in the pathogenesis of cardiovascular disease in type 2 diabetes
- (2015) Tushar P. Patel et al. HEART FAILURE REVIEWS
- Renal Lipotoxicity-Associated Inflammation and Insulin Resistance Affects Actin Cytoskeleton Organization in Podocytes
- (2015) Cristina Martínez-García et al. PLoS One
- Exercise Ameliorates Insulin Resistance via Ca2+Signals Distinct From Those of Insulin for GLUT4 Translocation in Skeletal Muscles
- (2014) Dae-Ryoung Park et al. DIABETES
- Non-receptor tyrosine kinases and the actin cytoskeleton in contractile vascular smooth muscle
- (2014) Jacqueline Ohanian et al. JOURNAL OF PHYSIOLOGY-LONDON
- MicroRNA-492 reverses high glucose-induced insulin resistance in HUVEC cells through targeting resistin
- (2014) Cai Ying et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Loss of neuronatin promotes “browning” of primary mouse adipocytes while reducing Glut1-mediated glucose disposal
- (2013) Valentina Gburcik et al. AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM
- Angiotensin II Promotes Thoracic Aortic Dissections and Ruptures in Col3a1 Haploinsufficient Mice
- (2013) Julie Faugeroux et al. HYPERTENSION
- Exercise Protects against Diet-Induced Insulin Resistance through Downregulation of Protein Kinase Cβ in Mice
- (2013) Xiaoquan Rao et al. PLoS One
- Disruption of Growth Factor Receptor-Binding Protein 10 in the Pancreas Enhances -Cell Proliferation and Protects Mice From Streptozotocin-Induced -Cell Apoptosis
- (2012) J. Zhang et al. DIABETES
- Regulation of Glucose Transport by ROCK1 Differs from That of ROCK2 and Is Controlled by Actin Polymerization
- (2012) Kwang-Hoon Chun et al. ENDOCRINOLOGY
- Rnd1 and Rnd3 targeting to lipid raft is required for p190 RhoGAP activation
- (2012) Izumi Oinuma et al. MOLECULAR BIOLOGY OF THE CELL
- TGF-β Receptor Signaling Pathways in Angiogenesis; Emerging Targets for Anti-Angiogenesis Therapy
- (2011) Laurens A. van Meeteren et al. CURRENT PHARMACEUTICAL BIOTECHNOLOGY
- Focal Adhesion Kinase Regulation of Mechanotransduction and its Impact on Endothelial Cell Functions
- (2011) Noureddine Zebda et al. MICROVASCULAR RESEARCH
- The role of Drosophila Merlin in spermatogenesis
- (2008) Natalia V Dorogova et al. BMC CELL BIOLOGY
- Use of Alternative Thresholds Defining Insulin Resistance to Predict Incident Type 2 Diabetes Mellitus and Cardiovascular Disease
- (2008) Martin K. Rutter et al. CIRCULATION
- Neuronatin: A New Inflammation Gene Expressed on the Aortic Endothelium of Diabetic Mice
- (2008) N. Mzhavia et al. DIABETES
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