Bclaf1 promotes angiogenesis by regulating HIF-1α transcription in hepatocellular carcinoma
出版年份 2018 全文链接
标题
Bclaf1 promotes angiogenesis by regulating HIF-1α transcription in hepatocellular carcinoma
作者
关键词
-
出版物
ONCOGENE
Volume -, Issue -, Pages -
出版商
Springer Nature America, Inc
发表日期
2018-10-26
DOI
10.1038/s41388-018-0552-1
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Platelet-derived microparticles promote endothelial cell proliferation in hypertension via miR-142-3p
- (2018) Han Bao et al. FASEB JOURNAL
- Hsp90α-dependent Bclaf1 promotes hepatocellular carcinoma proliferation by regulating c-MYC mRNA stability
- (2018) Xueqiong Zhou et al. HEPATOLOGY
- miR-194-5p/BCLAF1 deregulation in AML tumorigenesis
- (2017) C Dell'Aversana et al. LEUKEMIA
- The RNA processing factors THRAP3 and BCLAF1 promote the DNA damage response through selective mRNA splicing and nuclear export
- (2017) Jekaterina Vohhodina et al. NUCLEIC ACIDS RESEARCH
- Cancer statistics, 2016
- (2016) Rebecca L. Siegel et al. CA-A CANCER JOURNAL FOR CLINICIANS
- Bclaf1 is an important NF-κB signaling transducer and C/EBPβ regulator in DNA damage-induced senescence
- (2016) A-w Shao et al. CELL DEATH AND DIFFERENTIATION
- Nuclear respiratory factor-1 (NRF-1) regulated hypoxia-inducible factor-1α (HIF-1α) under hypoxia in HEK293T
- (2016) Dan Wang et al. IUBMB LIFE
- Hypoxic control of metastasis
- (2016) E. B. Rankin et al. SCIENCE
- Differentially Expressed miRNAs in Hepatocellular Carcinoma Target Genes in the Genetic Information Processing and Metabolism Pathways
- (2016) Thomas Thurnherr et al. Scientific Reports
- FBXO11 represses cellular response to hypoxia by destabilizing hypoxia-inducible factor-1α mRNA
- (2015) Uk-Il Ju et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
- STIM1 Mediates Hypoxia-Driven Hepatocarcinogenesis via Interaction with HIF-1
- (2015) Yongsheng Li et al. Cell Reports
- HIF-1α pathway: role, regulation and intervention for cancer therapy
- (2015) Georgina N. Masoud et al. Acta Pharmaceutica Sinica B
- HSP90 Supports Tumor Growth and Angiogenesis through PRKD2 Protein Stabilization
- (2014) N. Azoitei et al. CANCER RESEARCH
- Hypoxia inducible factors in liver disease and hepatocellular carcinoma: Current understanding and future directions
- (2014) Garrick K. Wilson et al. JOURNAL OF HEPATOLOGY
- Identification of a BRCA1-mRNA Splicing Complex Required for Efficient DNA Repair and Maintenance of Genomic Stability
- (2014) Kienan I. Savage et al. MOLECULAR CELL
- Tumor Cell Dissemination: Emerging Biological Insights from Animal Models and Cancer Patients
- (2013) Yibin Kang et al. CANCER CELL
- Btf and TRAP150 have distinct roles in regulating subcellular mRNA distribution
- (2013) Sapna Varia et al. Nucleus
- Epidemiology of Viral Hepatitis and Hepatocellular Carcinoma
- (2012) Hashem B. El-Serag GASTROENTEROLOGY
- MicroRNA target site polymorphisms in the VHL-HIF1α pathway predict renal cell carcinoma risk
- (2012) Hua Wei et al. MOLECULAR CARCINOGENESIS
- The cBio Cancer Genomics Portal: An Open Platform for Exploring Multidimensional Cancer Genomics Data: Figure 1.
- (2012) Ethan Cerami et al. Cancer Discovery
- Hallmarks of Cancer: The Next Generation
- (2011) Douglas Hanahan et al. CELL
- HCC and angiogenesis: possible targets and future directions
- (2011) Andrew X. Zhu et al. Nature Reviews Clinical Oncology
- In Search of a Function for BCLAF1
- (2011) Haya Sarras et al. TheScientificWorldJOURNAL
- Anchorage of VEGF to the extracellular matrix conveys differential signaling responses to endothelial cells
- (2010) Tom T. Chen et al. JOURNAL OF CELL BIOLOGY
- Targeting angiogenesis in hepatocellular carcinoma: focus on VEGF and bevacizumab
- (2009) Richard S Finn et al. Expert Review of Anticancer Therapy
- Essential role for Bclaf1 in lung development and immune system function
- (2008) J Peter McPherson et al. CELL DEATH AND DIFFERENTIATION
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