Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca
2+
/Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling
Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca
2+
/Calmodulin-Dependent Protein Kinase II δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling
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