UBQLN4 Represses Homologous Recombination and Is Overexpressed in Aggressive Tumors

出版年份 2019 全文链接

标题

作者

关键词

DNA damage, DNA double-strand break repair, homologous recombination, non-homologous end joining, cancer, targeted cancer therapy, UBQLN4 deficiency syndrome, genome instability syndrome, ubiquitin, proteasomal degradation

出版物

CELL

Volume -, Issue -, Pages -

出版商

Elsevier BV

发表日期

2019-01-04

DOI

10.1016/j.cell.2018.11.024

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