MicroRNA Expression Profile of Pulmonary Artery Smooth Muscle Cells and the Effect of Let-7d in Chronic Thromboembolic Pulmonary Hypertension

Effects of let-7 microRNA on Cell Growth and Differentiation of Papillary Thyroid Cancer

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p53 Gene deficiency promotes hypoxia-induced pulmonary hypertension and vascular remodeling in mice

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Vascular Smooth Muscle Cell Proliferation Is Influenced by let-7d MicroRNA and Its Interaction With KRAS

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Role for miR-204 in human pulmonary arterial hypertension

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Mechanisms of disease: pulmonary arterial hypertension

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MicroRNA-21 plays a role in hypoxia-mediated pulmonary artery smooth muscle cell proliferation and migration

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Dynamic Changes in Lung MicroRNA Profiles During the Development of Pulmonary Hypertension due to Chronic Hypoxia and Monocrotaline

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miRNAs: roles and clinical applications in vascular disease

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Susceptibility to chronic thromboembolic pulmonary hypertension may be conferred by miR-759 via its targeted interaction with polymorphic fibrinogen alpha gene

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Tumor suppressor microRNAs are underrepresented in primary effusion lymphoma and Kaposi sarcoma

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Interleukin-6 Modulates the Expression of the Bone Morphogenic Protein Receptor Type II Through a Novel STAT3–microRNA Cluster 17/92 Pathway

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Let-7a elevates p21WAF1levels by targeting of NIRF and suppresses the growth of A549 lung cancer cells

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let-7Overexpression Leads to an Increased Fraction of Cells in G2/M, Direct Down-regulation of Cdc34, and Stabilization of Wee1 Kinase in Primary Fibroblasts

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Hypoxia-Inducible mir-210 Regulates Normoxic Gene Expression Involved in Tumor Initiation

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Molecular pathogenesis of pulmonary arterial hypertension

(2008) Marlene Rabinovitch   JOURNAL OF CLINICAL INVESTIGATION

Widespread changes in protein synthesis induced by microRNAs

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The expanding world of small RNAs

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Suppression of non-small cell lung tumor development by the let-7 microRNA family

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