Role of Protein Kinase C in Podocytes and Development of Glomerular Damage in Diabetic Nephropathy
出版年份 2014 全文链接
标题
Role of Protein Kinase C in Podocytes and Development of Glomerular Damage in Diabetic Nephropathy
作者
关键词
-
出版物
Frontiers in Endocrinology
Volume 5, Issue -, Pages -
出版商
Frontiers Media SA
发表日期
2014-11-05
DOI
10.3389/fendo.2014.00179
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- aPKCλ maintains the integrity of the glomerular slit diaphragm through trafficking of nephrin to the cell surface
- (2014) Daisuke Satoh et al. JOURNAL OF BIOCHEMISTRY
- Def-6, a Novel Regulator of Small GTPases in Podocytes, Acts Downstream of Atypical Protein Kinase C (aPKC) λ/ι
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- Dual Inhibition of Classical Protein Kinase C- and Protein Kinase C- Isoforms Protects Against Experimental Murine Diabetic Nephropathy
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- aPKC / and aPKC Contribute to Podocyte Differentiation and Glomerular Maturation
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- Podocytopathy in Diabetes: A Metabolic and Endocrine Disorder
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- Overexpression of VEGF-A in podocytes of adult mice causes glomerular disease
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- Podocytic PKC-Alpha Is Regulated in Murine and Human Diabetes and Mediates Nephrin Endocytosis
- (2010) Irini Tossidou et al. PLoS One
- PKC-alpha Modulates TGF-β Signaling and Impairs Podocyte Survival
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- Loss of Podocyte aPKC / Causes Polarity Defects and Nephrotic Syndrome
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- Wnt/ -Catenin Signaling Promotes Podocyte Dysfunction and Albuminuria
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- Targeting PKC: a novel role for beta-catenin in ER stress and apoptotic signaling
- (2008) M. S. Raab et al. BLOOD
- The kidney in diabetes: dynamic pathways of injury and repair. The Camillo Golgi Lecture 2007
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- Neph-Nephrin Proteins Bind the Par3-Par6-Atypical Protein Kinase C (aPKC) Complex to Regulate Podocyte Cell Polarity
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- Plasma membrane recruitment of dephosphorylated -catenin upon activation of the Wnt pathway
- (2008) J. Hendriksen et al. JOURNAL OF CELL SCIENCE
- Structural Basis of Protein Kinase C Isoform Function
- (2008) Susan F. Steinberg PHYSIOLOGICAL REVIEWS
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