Genetic disruption of Ano5 in mice does not recapitulate human ANO5-deficient muscular dystrophy

Down-regulation of LXR/RXR activation and negative acute phase response pathways in colon adenocarcinoma revealed by proteomics and bioinformatics analysis

(2017) Hui Tang et al.   Cancer Biomarkers

A comparative encyclopedia of DNA elements in the mouse genome

(2014) Feng Yue et al.   NATURE

Prednisolone Attenuates Improvement of Cardiac and Skeletal Contractile Function and Histopathology by Lisinopril and Spironolactone in the mdx Mouse Model of Duchenne Muscular Dystrophy

(2014) Paul M. L. Janssen et al.   PLoS One

Anoctamin 6 Regulates C2C12 Myoblast Proliferation

(2014) Piming Zhao et al.   PLoS One

TMEM16F is a component of a Ca2+-activated Cl− channel but not a volume-sensitive outwardly rectifying Cl− channel

(2013) Takahiro Shimizu et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

ANO5-muscular dystrophy: clinical, pathological and molecular findings

(2013) T. Liewluck et al.   EUROPEAN JOURNAL OF NEUROLOGY

ANO5Gene Analysis in a Large Cohort of Patients with Anoctaminopathy: Confirmation of Male Prevalence and High Occurrence of the Common Exon 5 Gene Mutation

(2013) Anna Sarkozy et al.   HUMAN MUTATION

Anoctamin 5 muscular dystrophy in Denmark: prevalence, genotypes, phenotypes, cardiac findings, and muscle protein expression

(2013) Nanna Witting et al.   JOURNAL OF NEUROLOGY

Purified TMEM16A is sufficient to form Ca2+-activated Cl- channels

(2013) H. Terashima et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signaling

(2013) Adrian Britschgi et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Inhibition of Ca2+-activated Cl− channel ANO1/TMEM16A expression suppresses tumor growth and invasiveness in human prostate carcinoma

(2012) Wen Liu et al.   CANCER LETTERS

TMEM16A Induces MAPK and Contributes Directly to Tumorigenesis and Cancer Progression

(2012) U. Duvvuri et al.   CANCER RESEARCH

TMEM16F Forms a Ca2+-Activated Cation Channel Required for Lipid Scrambling in Platelets during Blood Coagulation

(2012) Huanghe Yang et al.   CELL

A novel missense mutation in ANO5/TMEM16E is causative for gnathodiaphyseal dyplasia in a large Italian pedigree

(2012) Caterina Marconi et al.   EUROPEAN JOURNAL OF HUMAN GENETICS

Dilated cardiomyopathy in patients with mutations in anoctamin 5

(2012) K. Wahbi et al.   INTERNATIONAL JOURNAL OF CARDIOLOGY

Myopathy caused by anoctamin 5 mutations and necrotizing vasculitis

(2012) Isabelle Pénisson-Besnier et al.   JOURNAL OF NEUROLOGY

Novel mutations in the anoctamin 5 gene (ANO5) associated with limb-girdle muscular dystrophy 2L

(2012) Ann A. Little et al.   MUSCLE & NERVE

Novel ANO5 mutations causing hyper-CK-emia, limb girdle muscular weakness and miyoshi type of muscular dystrophy

(2012) Joachim Schessl et al.   MUSCLE & NERVE

Frequency and characterisation of anoctamin 5 mutations in a cohort of Italian limb-girdle muscular dystrophy patients

(2012) Francesca Magri et al.   NEUROMUSCULAR DISORDERS

Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction

(2012) F. Huang et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Overexpression, crystallization and preliminary X-ray crystallographic analysis of the C-terminal cytosolic domain of mouse anoctamin 1

(2011) Sang Ho Park et al.   Acta Crystallographica Section F-Structural Biology and Crystallization Communications

Physiological roles and diseases of tmem16/anoctamin proteins: are they all chloride channels?

(2011) Charity Duran et al.   ACTA PHARMACOLOGICA SINICA

ANOs 3–7 in the anoctamin/Tmem16 Cl− channel family are intracellular proteins

(2011) Charity Duran et al.   AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY

Dystrophin deficiency exacerbates skeletal muscle pathology in dysferlin-null mice

(2011) Renzhi Han et al.   Skeletal Muscle

Recessive Mutations in the Putative Calcium-Activated Chloride Channel Anoctamin 5 Cause Proximal LGMD2L and Distal MMD3 Muscular Dystrophies

(2010) Véronique Bolduc et al.   AMERICAN JOURNAL OF HUMAN GENETICS

A founder mutation in Anoctamin 5 is a major cause of limb girdle muscular dystrophy

(2010) D. Hicks et al.   BRAIN

Expression and Function of Epithelial Anoctamins

(2010) Rainer Schreiber et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Genetic ablation of complement C3 attenuates muscle pathology in dysferlin-deficient mice

(2010) Renzhi Han et al.   JOURNAL OF CLINICAL INVESTIGATION

Retinoic acid represses CYP7A1 expression in human hepatocytes and HepG2 cells by FXR/RXR-dependent and independent mechanisms

(2010) Shi-Ying Cai et al.   JOURNAL OF LIPID RESEARCH

TMEM16A/Anoctamin 1 protein mediates calcium-activated chloride currents in pulmonary arterial smooth muscle cells

(2010) Boris Manoury et al.   JOURNAL OF PHYSIOLOGY-LONDON

Basal lamina strengthens cell membrane integrity via the laminin G domain-binding motif of  -dystroglycan

(2009) R. Han et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

TMEM16A confers receptor-activated calcium-dependent chloride conductance

(2008) Young Duk Yang et al.   NATURE

TMEM16A, A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity

(2008) A. Caputo et al.   SCIENCE