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Molecular, Cellular and Structural Mechanisms of Cocaine Addiction: A Key Role for MicroRNAs

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FosB Is Essential for the Enhancement of Stress Tolerance and Antagonizes Locomotor Sensitization by ΔFosB

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Intronic Polymorphisms Affecting Alternative Splicing of Human Dopamine D2 Receptor Are Associated with Cocaine Abuse

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Essential Role of the Histone Methyltransferase G9a in Cocaine-Induced Plasticity

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Modulation of Chromatin Modification Facilitates Extinction of Cocaine-Induced Conditioned Place Preference

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An inducible change in Fox-1/A2BP1 splicing modulates the alternative splicing of downstream neuronal target exons

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Genome-wide Analysis of Chromatin Regulation by Cocaine Reveals a Role for Sirtuins

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