PU.1 target genes undergo Tet2-coupled demethylation and DNMT3b-mediated methylation in monocyte-to-osteoclast differentiation

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Stage-Specific Roles for Tet1 and Tet2 in DNA Demethylation in Primordial Germ Cells

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5-Hydroxymethylcytosine is an essential intermediate of active DNA demethylation processes in primary human monocytes

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The B cell transcription program mediates hypomethylation and overexpression of key genes in Epstein-Barr virus-associated proliferative conversion

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Dynamic epigenetic enhancer signatures reveal key transcription factors associated with monocytic differentiation states

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Dynamic Transformations of Genome-wide Epigenetic Marking and Transcriptional Control Establish T Cell Identity

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Genome-wide DNA methylation profiles in hematopoietic stem and progenitor cells reveal overrepresentation of ETS transcription factor binding sites

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Tet2 Facilitates the Derepression of Myeloid Target Genes during CEBPα-Induced Transdifferentiation of Pre-B Cells

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Bach1 regulates osteoclastogenesis in a mouse model via both heme oxygenase 1-dependent and heme oxygenase 1-independent pathways

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Thymine DNA Glycosylase Is Essential for Active DNA Demethylation by Linked Deamination-Base Excision Repair

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Tet Proteins Can Convert 5-Methylcytosine to 5-Formylcytosine and 5-Carboxylcytosine

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Transcriptionally active nuclei are selective in mature multinucleated osteoclasts

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Transcription factor C/EBPβ isoform ratio regulates osteoclastogenesis through MafB

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Ca2+-NFATc1 signaling is an essential axis of osteoclast differentiation

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Inhibitory ITAMs as novel regulators of immunity

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Complex Regulation of Tartrate-resistant Acid Phosphatase (TRAP) Expression by Interleukin 4 (IL-4)

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Genome-wide expression analyses establish dendritic cells as a new osteoclast precursor able to generate bone-resorbing cells more efficiently than monocytes

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Conversion of 5-Methylcytosine to 5-Hydroxymethylcytosine in Mammalian DNA by MLL Partner TET1

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The Nuclear DNA Base 5-Hydroxymethylcytosine Is Present in Purkinje Neurons and the Brain

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Actin cytoskeletal organisation in osteoclasts: A model to decipher transmigration and matrix degradation

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