标题
The central role of EED in the orchestration of polycomb group complexes
作者
关键词
-
出版物
Nature Communications
Volume 5, Issue 1, Pages -
出版商
Springer Nature
发表日期
2014-01-24
DOI
10.1038/ncomms4127
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Fbxl10/Kdm2b Recruits Polycomb Repressive Complex 1 to CpG Islands and Regulates H2A Ubiquitylation
- (2013) Xudong Wu et al. MOLECULAR CELL
- Kdm2b maintains murine embryonic stem cell status by recruiting PRC1 complex to CpG islands of developmental genes
- (2013) Jin He et al. NATURE CELL BIOLOGY
- RYBP-PRC1 Complexes Mediate H2A Ubiquitylation at Polycomb Target Sites Independently of PRC2 and H3K27me3
- (2012) Lígia Tavares et al. CELL
- PCGF Homologs, CBX Proteins, and RYBP Define Functionally Distinct PRC1 Family Complexes
- (2012) Zhonghua Gao et al. MOLECULAR CELL
- Characterization of the EZH2-MMSET Histone Methyltransferase Regulatory Axis in Cancer
- (2012) Irfan A. Asangani et al. MOLECULAR CELL
- EZH2 inhibition as a therapeutic strategy for lymphoma with EZH2-activating mutations
- (2012) Michael T. McCabe et al. NATURE
- Histone methylation: a dynamic mark in health, disease and inheritance
- (2012) Eric L. Greer et al. NATURE REVIEWS GENETICS
- REST–Mediated Recruitment of Polycomb Repressor Complexes in Mammalian Cells
- (2012) Nikolaj Dietrich et al. PLoS Genetics
- Molecular architecture of human polycomb repressive complex 2
- (2012) Claudio Ciferri et al. eLife
- KDM2B links the Polycomb Repressive Complex 1 (PRC1) to recognition of CpG islands
- (2012) Anca M Farcas et al. eLife
- Coordinated Regulation of Polycomb Group Complexes through microRNAs in Cancer
- (2011) Qi Cao et al. CANCER CELL
- Mechanistic Rationale for Inhibition of Poly(ADP-Ribose) Polymerase in ETS Gene Fusion-Positive Prostate Cancer
- (2011) J. Chad Brenner et al. CANCER CELL
- Cancer epigenetics: linking basic biology to clinical medicine
- (2011) Hsing-Chen Tsai et al. CELL RESEARCH
- The Polycomb complex PRC2 and its mark in life
- (2011) Raphaël Margueron et al. NATURE
- Abacus: A computational tool for extracting and pre-processing spectral count data for label-free quantitative proteomic analysis
- (2011) Damian Fermin et al. PROTEOMICS
- Jarid2 and PRC2, partners in regulating gene expression
- (2010) G. Li et al. GENES & DEVELOPMENT
- Polycomb complexes act redundantly to repress genomic repeats and genes
- (2010) M. Leeb et al. GENES & DEVELOPMENT
- JARID2 regulates binding of the Polycomb repressive complex 2 to target genes in ES cells
- (2010) Diego Pasini et al. NATURE
- Jarid2 is a PRC2 component in embryonic stem cells required for multi-lineage differentiation and recruitment of PRC1 and RNA Polymerase II to developmental regulators
- (2010) David Landeira et al. NATURE CELL BIOLOGY
- Somatic mutations altering EZH2 (Tyr641) in follicular and diffuse large B-cell lymphomas of germinal-center origin
- (2010) Ryan D Morin et al. NATURE GENETICS
- Fast and accurate short read alignment with Burrows-Wheeler transform
- (2009) H. Li et al. BIOINFORMATICS
- Jarid2/Jumonji Coordinates Control of PRC2 Enzymatic Activity and Target Gene Occupancy in Pluripotent Cells
- (2009) Jamy C. Peng et al. CELL
- Jumonji Modulates Polycomb Activity and Self-Renewal versus Differentiation of Stem Cells
- (2009) Xiaohua Shen et al. CELL
- Role of the polycomb protein EED in the propagation of repressive histone marks
- (2009) Raphael Margueron et al. NATURE
- In Brief
- (2009) NATURE REVIEWS NEUROSCIENCE
- Cooperation between EZH2, NSPc1-mediated histone H2A ubiquitination and Dnmt1 in HOX gene silencing
- (2008) Xudong Wu et al. NUCLEIC ACIDS RESEARCH
- Genomic Loss of microRNA-101 Leads to Overexpression of Histone Methyltransferase EZH2 in Cancer
- (2008) S. Varambally et al. SCIENCE
- Genomewide Analysis of PRC1 and PRC2 Occupancy Identifies Two Classes of Bivalent Domains
- (2008) Manching Ku et al. PLoS Genetics
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