The bHLH transcription factor DEC1 promotes thyroid cancer aggressiveness by the interplay with NOTCH1
出版年份 2018 全文链接
标题
The bHLH transcription factor DEC1 promotes thyroid cancer aggressiveness by the interplay with NOTCH1
作者
关键词
-
出版物
Cell Death & Disease
Volume 9, Issue 9, Pages -
出版商
Springer Nature America, Inc
发表日期
2018-08-29
DOI
10.1038/s41419-018-0933-y
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- The role of the Hes1 crosstalk hub in Notch-Wnt interactions of the intestinal crypt
- (2017) Sophie K. Kay et al. PLoS Computational Biology
- Notch1 Signaling Regulates the Aggressiveness of Differentiated Thyroid Cancer and Inhibits SERPINE1 Expression
- (2016) X.-M. Yu et al. CLINICAL CANCER RESEARCH
- DEC1 and DEC2 Crosstalk between Circadian Rhythm and Tumor Progression
- (2016) Fuyuki Sato et al. Journal of Cancer
- Basic helix-loop-helix transcription factor DEC1 regulates the cisplatin-induced apoptotic pathway of human esophageal cancer cells
- (2015) Hiroko SEINO et al. BIOMEDICAL RESEARCH-TOKYO
- A Classification of Basic Helix-Loop-Helix Transcription Factors of Soybean
- (2015) Karen A. Hudson et al. International Journal of Genomics
- Time to re-consider the meaning of BRAF V600E mutation in papillary thyroid carcinoma
- (2014) Greta Gandolfi et al. INTERNATIONAL JOURNAL OF CANCER
- DEC1 Coordinates with HDAC8 to Differentially Regulate TAp73 and ΔNp73 Expression
- (2014) Yingjuan Qian et al. PLoS One
- Notch Pathway Is Activated by MAPK Signaling and Influences Papillary Thyroid Cancer Proliferation
- (2014) Alex Shimura Yamashita et al. Translational Oncology
- Update on Anaplastic Thyroid Carcinoma: Morphological, Molecular, and Genetic Features of the Most Aggressive Thyroid Cancer
- (2014) Moira Ragazzi et al. International Journal of Endocrinology
- Nontranscriptional Role of Hif-1α in Activation of γ-Secretase and Notch Signaling in Breast Cancer
- (2014) Jennifer C. Villa et al. Cell Reports
- Hypoxia-induced overexpression of DEC1 is regulated by HIF-1α in hepatocellular carcinoma
- (2013) WANSHAN MA et al. ONCOLOGY REPORTS
- BHLH transcription factor DEC2 regulates pro-apoptotic factor Bim in human oral cancer HSC-3 cells
- (2012) Yunyan Wu et al. BIOMEDICAL RESEARCH-TOKYO
- Runx2 Isoform I Controls a Panel of Proinvasive Genes Driving Aggressiveness of Papillary Thyroid Carcinomas
- (2012) Valentina Sancisi et al. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM
- Overexpression of the DEC1 Protein Induces Senescence In Vitro and Is Related to Better Survival in Esophageal Squamous Cell Carcinoma
- (2012) Qing Xu et al. PLoS One
- HIF-1α is critical for hypoxia-mediated maintenance of glioblastoma stem cells by activating Notch signaling pathway
- (2011) L Qiang et al. CELL DEATH AND DIFFERENTIATION
- Thyroid Cancer Survival in the United States
- (2010) Louise Davies et al. ARCHIVES OF OTOLARYNGOLOGY-HEAD & NECK SURGERY
- Inhibitor of DNA binding-1 induces mesenchymal features and promotes invasiveness in thyroid tumour cells
- (2010) Alessia Ciarrocchi et al. EUROPEAN JOURNAL OF CANCER
- Notch Activation Induces Akt Signaling via an Autocrine Loop to Prevent Apoptosis in Breast Epithelial Cells
- (2009) O. Meurette et al. CANCER RESEARCH
- The Notch signaling pathway: Transcriptional regulation at Notch target genes
- (2009) T. Borggrefe et al. CELLULAR AND MOLECULAR LIFE SCIENCES
- Notch1 mediates growth suppression of papillary and follicular thyroid cancer cells by histone deacetylase inhibitors
- (2009) X. Xiao et al. MOLECULAR CANCER THERAPEUTICS
- The Hypoxia-Regulated Transcription Factor DEC1 (Stra13, SHARP-2) and Its Expression in Gastric Cancer
- (2009) Yan Zheng et al. OMICS-A JOURNAL OF INTEGRATIVE BIOLOGY
- ID1, Inhibitor of Differentiation/DNA Binding, Is an Effector of the p53-dependent DNA Damage Response Pathway
- (2008) Yingjuan Qian et al. JOURNAL OF BIOLOGICAL CHEMISTRY
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