期刊
VIRUSES-BASEL
卷 3, 期 4, 页码 293-311出版社
MDPI
DOI: 10.3390/v3040293
关键词
HIV; actin cytoskeleton; entry; Nef; cofilin
类别
资金
- Deutsche Forschungsgemeinschaft [SFB 638, GRK1188]
The host cell cytoskeleton plays a key role in the life cycle of viral pathogens whose propagation depends on mandatory intracellular steps. Accordingly, also the human immunodeficiency virus type 1 (HIV-1) has evolved strategies to exploit and modulate in particular the actin cytoskeleton for its purposes. This review will recapitulate recent findings on how HIV-1 hijacks the cytoskeleton to facilitate entry into, transport within and egress from host cells as well as to commandeer communication of infected with uninfected bystander cells.
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