Skeletal defects in Osterix-Cre transgenic mice

Osx-Cre Targets Multiple Cell Types besides Osteoblast Lineage in Postnatal Mice

(2014) Jianquan Chen et al.   PLoS One

In vivo expression of Osterix in mature granule cells of adult mouse olfactory bulb

(2011) Ji-Soo Park et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Conditional Inactivation of the CXCR4 Receptor in Osteoprecursors Reduces Postnatal Bone Formation Due to Impaired Osteoblast Development

(2011) Wei Zhu et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

GαqSignal in Osteoblasts Is Inhibitory to the Osteoanabolic Action of Parathyroid Hormone

(2011) Naoshi Ogata et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Decreased body weight in young Osterix-Cre transgenic mice results in delayed cortical bone expansion and accrual

(2011) Rachel A. Davey et al.   TRANSGENIC RESEARCH

Genetic and pathologic aspects of retinoic acid-induced limb malformations in the mouse

(2010) Grace S. Lee et al.   BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY

Glucocorticoids Suppress Bone Formation by Attenuating Osteoblast Differentiation via the Monomeric Glucocorticoid Receptor

(2010) Alexander Rauch et al.   Cell Metabolism

The p38 MAPK pathway is essential for skeletogenesis and bone homeostasis in mice

(2010) Matthew B. Greenblatt et al.   JOURNAL OF CLINICAL INVESTIGATION

Histone Deacetylase 3 Depletion in Osteo/Chondroprogenitor Cells Decreases Bone Density and Increases Marrow Fat

(2010) David F. Razidlo et al.   PLoS One

BMP signaling negatively regulates bone mass through sclerostin by inhibiting the canonical Wnt pathway

(2008) N. Kamiya et al.   DEVELOPMENT

A mouse model for polycystic kidney disease through a somatic in-frame deletion in the 5′ end of Pkd1

(2008) P.G. Starremans et al.   KIDNEY INTERNATIONAL