期刊
SCIENCE SIGNALING
卷 11, 期 549, 页码 -出版社
AMER ASSOC ADVANCEMENT SCIENCE
DOI: 10.1126/scisignal.aav1646
关键词
-
资金
- NEI NIH HHS [R01 EY011500] Funding Source: Medline
- NIDA NIH HHS [R21 DA030103] Funding Source: Medline
- NIGMS NIH HHS [R35 GM122491] Funding Source: Medline
- NINDS NIH HHS [R01 NS065868] Funding Source: Medline
Several studies have suggested that arrestin-mediated signaling by GPCRs requires G protein activation; however, in this issue of Science Signaling, Luttrell et al. documented arrestin-dependent activation of ERK1/2 by a number of GPCRs. These studies do not contradict each other, but illustrate the complexity of cellular signaling that cannot and should not be reduced to simplistic models.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据