Inhibition of Autophagy Ameliorates Acute Lung Injury Caused by Avian Influenza A H5N1 Infection

The role of TOR in autophagy regulation from yeast to plants and mammals

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A functionalized single-walled carbon nanotube-induced autophagic cell death in human lung cells through Akt–TSC2-mTOR signaling

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Autophagy is involved in influenza A virus replication

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Matrix Protein 2 of Influenza A Virus Blocks Autophagosome Fusion with Lysosomes

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H5N1 influenza viruses: outbreaks and biological properties

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PAMAM Nanoparticles Promote Acute Lung Injury by Inducing Autophagic Cell Death through the Akt-TSC2-mTOR Signaling Pathway

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Avian influenza H5N1: an update on molecular pathogenesis

(2009) HongLiang Wang et al.   Science in China. Series C, Life sciences / Chinese Academy of Sciences

Influenza A virus H5N1 entry into host cells is through clathrin-dependent endocytosis

(2009) HongLiang Wang et al.   Science in China. Series C, Life sciences / Chinese Academy of Sciences

Pathology, Molecular Biology, and Pathogenesis of Avian Influenza A (H5N1) Infection in Humans

(2008) Christine Korteweg et al.   AMERICAN JOURNAL OF PATHOLOGY

Identification of Oxidative Stress and Toll-like Receptor 4 Signaling as a Key Pathway of Acute Lung Injury

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Human H5N1 influenza: Current insight into pathogenesis

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Synergistic anti‐proliferative and pro‐apoptotic activity of combined therapy with bortezomib, a proteasome inhibitor, with anti‐epidermal growth factor receptor (EGFR) drugs in human cancer cells

(2008) Tina Cascone et al.   JOURNAL OF CELLULAR PHYSIOLOGY

Generation and characterization of an H5N1 avian influenza virus hemagglutinin glycoprotein pseudotyped lentivirus

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Human infection with highly pathogenic H5N1 influenza virus

(2008) Andrea Gambotto et al.   LANCET

Clinical Characteristics of 26 Human Cases of Highly Pathogenic Avian Influenza A (H5N1) Virus Infection in China

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Denbinobin induces apoptosis in human lung adenocarcinoma cells via Akt inactivation, Bad activation, and mitochondrial dysfunction

(2008) Chen-Tzu Kuo et al.   TOXICOLOGY LETTERS