Decreased cathepsin V expression due to Fli1 deficiency contributes to the development of dermal fibrosis and proliferative vasculopathy in systemic sclerosis
出版年份 2013 全文链接
标题
Decreased cathepsin V expression due to Fli1 deficiency contributes to the development of dermal fibrosis and proliferative vasculopathy in systemic sclerosis
作者
关键词
-
出版物
RHEUMATOLOGY
Volume 52, Issue 5, Pages 790-799
出版商
Oxford University Press (OUP)
发表日期
2013-01-04
DOI
10.1093/rheumatology/kes379
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- A Possible Contribution of Altered Cathepsin B Expression to the Development of Skin Sclerosis and Vasculopathy in Systemic Sclerosis
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- Endothelial Fli1 Deficiency Impairs Vascular Homeostasis
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- Phosphorylation of Fli1 at Threonine 312 by Protein Kinase C Promotes Its Interaction with p300/CREB-Binding Protein-Associated Factor and Subsequent Acetylation in Response to Transforming Growth Factor
- (2009) Y. Asano et al. MOLECULAR AND CELLULAR BIOLOGY
- Angiogenesis and its targeting in rheumatoid arthritis
- (2009) Zoltán Szekanecz et al. VASCULAR PHARMACOLOGY
- Extracellular catalase activity protects cysteine cathepsins from inactivation by hydrogen peroxide
- (2008) Virginie Hervé-Grépinet et al. FEBS LETTERS
- Proteomic analysis of scleroderma lesional skin reveals activated wound healing phenotype of epidermal cell layer
- (2008) N. Aden et al. RHEUMATOLOGY
- Cathepsin V, but not cathepsins L, B and K, may release angiostatin-like fragments from plasminogen
- (2007) Luciano Puzer et al. BIOLOGICAL CHEMISTRY
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