Dietary sugar promotes systemic TOR activation in Drosophila through AKH-dependent selective secretion of Dilp3
出版年份 2015 全文链接
标题
Dietary sugar promotes systemic TOR activation in Drosophila through AKH-dependent selective secretion of Dilp3
作者
关键词
-
出版物
Nature Communications
Volume 6, Issue 1, Pages -
出版商
Springer Nature
发表日期
2015-04-17
DOI
10.1038/ncomms7846
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Sucrose Induces Vesicle Accumulation and Autophagy
- (2015) Takahiro Higuchi et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Modeling metabolic homeostasis and nutrient sensing in Drosophila: implications for aging and metabolic diseases
- (2014) E. Owusu-Ansah et al. Disease Models & Mechanisms
- Methods for studying metabolism in Drosophila
- (2014) Jason M. Tennessen et al. METHODS
- Autophagy—a key player in cellular and body metabolism
- (2014) Kook Hwan Kim et al. Nature Reviews Endocrinology
- Eosinophil-derived cytokines in health and disease: unraveling novel mechanisms of selective secretion
- (2013) R. C. N. Melo et al. ALLERGY
- Metabolic Signaling in Fuel-Induced Insulin Secretion
- (2013) Marc Prentki et al. Cell Metabolism
- A secreted decoy of InR antagonizes insulin/IGF signaling to restrict body growth in Drosophila
- (2013) N. Okamoto et al. GENES & DEVELOPMENT
- Drosophila Adiponectin Receptor in Insulin Producing Cells Regulates Glucose and Lipid Metabolism by Controlling Insulin Secretion
- (2013) Su-Jin Kwak et al. PLoS One
- Functional implications of Drosophila insulin-like peptides in metabolism, aging, and dietary restriction
- (2013) Kavitha Kannan et al. Frontiers in Physiology
- Drosophila Cytokine Unpaired 2 Regulates Physiological Homeostasis by Remotely Controlling Insulin Secretion
- (2012) Akhila Rajan et al. CELL
- Unique roles of glucagon and glucagon-like peptides: Parallels in understanding the functions of adipokinetic hormones in stress responses in insects
- (2012) Andrea Bednářová et al. COMPARATIVE BIOCHEMISTRY AND PHYSIOLOGY A-MOLECULAR & INTEGRATIVE PHYSIOLOGY
- Role of mammalian homologue of Caenorhabditis elegans unc-13-1 (Munc13-1) in the recruitment of newcomer insulin granules in both first and second phases of glucose-stimulated insulin secretion in mouse islets
- (2012) L. Xie et al. DIABETOLOGIA
- Energy-Dependent Modulation of Glucagon-Like Signaling in Drosophila via the AMP-Activated Protein Kinase
- (2012) J. T. Braco et al. GENETICS
- Temporal Coding of Insulin Action through Multiplexing of the AKT Pathway
- (2012) Hiroyuki Kubota et al. MOLECULAR CELL
- High Sugar-Induced Insulin Resistance in Drosophila Relies on the Lipocalin Neural Lazarillo
- (2012) Matthieu Y. Pasco et al. PLoS One
- A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila
- (2011) L. Palanker Musselman et al. Disease Models & Mechanisms
- Molecular Evolution and Functional Characterization of Drosophila Insulin-Like Peptides
- (2010) Sebastian Grönke et al. PLoS Genetics
- Remote Control of Insulin Secretion by Fat Cells in Drosophila
- (2009) Charles Géminard et al. Cell Metabolism
- An Atg1/Atg13 Complex with Multiple Roles in TOR-mediated Autophagy Regulation
- (2009) Yu-Yun Chang et al. MOLECULAR BIOLOGY OF THE CELL
- Deletion of Drosophila insulin-like peptides causes growth defects and metabolic abnormalities
- (2009) H. Zhang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Regulatory peptides in fruit fly midgut
- (2008) Jan A. Veenstra et al. CELL AND TISSUE RESEARCH
- Drosophila ALS Regulates Growth and Metabolism through Functional Interaction with Insulin-Like Peptides
- (2008) Nathalie Arquier et al. Cell Metabolism
- Opposing Effects of Dietary Protein and Sugar Regulate a Transcriptional Target of Drosophila Insulin-like Peptide Signaling
- (2008) Susanne Buch et al. Cell Metabolism
Become a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get StartedAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started