MicroRNA-431 accelerates muscle regeneration and ameliorates muscular dystrophy by targeting Pax7 in mice
出版年份 2015 全文链接
标题
MicroRNA-431 accelerates muscle regeneration and ameliorates muscular dystrophy by targeting Pax7 in mice
作者
关键词
-
出版物
Nature Communications
Volume 6, Issue 1, Pages -
出版商
Springer Nature
发表日期
2015-07-07
DOI
10.1038/ncomms8713
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Pax7 is critical for the normal function of satellite cells in adult skeletal muscle
- (2013) J. von Maltzahn et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A Subpopulation of Adult Skeletal Muscle Stem Cells Retains All Template DNA Strands after Cell Division
- (2012) Pierre Rocheteau et al. CELL
- HMGB1-RAGE regulates muscle satellite cell homeostasis through p38-MAPK- and myogenin-dependent repression of Pax7 transcription
- (2012) F. Riuzzi et al. JOURNAL OF CELL SCIENCE
- microRNA-206 promotes skeletal muscle regeneration and delays progression of Duchenne muscular dystrophy in mice
- (2012) Ning Liu et al. JOURNAL OF CLINICAL INVESTIGATION
- Maintenance of muscle stem-cell quiescence by microRNA-489
- (2012) Tom H. Cheung et al. NATURE
- Wnt7a treatment ameliorates muscular dystrophy
- (2012) J. von Maltzahn et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Pax7-expressing satellite cells are indispensable for adult skeletal muscle regeneration
- (2011) R. Sambasivan et al. DEVELOPMENT
- An absolute requirement for Pax7-positive satellite cells in acute injury-induced skeletal muscle regeneration
- (2011) C. Lepper et al. DEVELOPMENT
- Regulation of DMD pathology by an ankyrin-encoded miRNA
- (2011) Matthew S Alexander et al. Skeletal Muscle
- Matrix Metalloproteinase Inhibitor Batimastat Alleviates Pathology and Improves Skeletal Muscle Function in Dystrophin-Deficient mdx Mice
- (2010) Akhilesh Kumar et al. AMERICAN JOURNAL OF PATHOLOGY
- MicroRNAs Involved in Molecular Circuitries Relevant for the Duchenne Muscular Dystrophy Pathogenesis Are Controlled by the Dystrophin/nNOS Pathway
- (2010) Davide Cacchiarelli et al. Cell Metabolism
- microRNA-1 and microRNA-206 regulate skeletal muscle satellite cell proliferation and differentiation by repressing Pax7
- (2010) Jian-Fu Chen et al. JOURNAL OF CELL BIOLOGY
- miR-206 and -486 Induce Myoblast Differentiation by Downregulating Pax7
- (2010) B. K. Dey et al. MOLECULAR AND CELLULAR BIOLOGY
- The Imprinted Retrotransposon-Like Gene PEG11 (RTL1) Is Expressed as a Full-Length Protein in Skeletal Muscle from Callipyge Sheep
- (2010) Keren Byrne et al. PLoS One
- Matrix metalloproteinase-9 inhibition ameliorates pathogenesis and improves skeletal muscle regeneration in muscular dystrophy
- (2009) H. Li et al. HUMAN MOLECULAR GENETICS
- MicroRNA-1/206 Targets c-Metand Inhibits Rhabdomyosarcoma Development
- (2009) Dongsheng Yan et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Adult satellite cells and embryonic muscle progenitors have distinct genetic requirements
- (2009) Christoph Lepper et al. NATURE
- At Least Ten Genes Define the Imprinted Dlk1-Dio3 Cluster on Mouse Chromosome 12qF1
- (2009) John P. Hagan et al. PLoS One
- Muscle stem cell behavior is modified by microRNA-27 regulation of Pax3 expression
- (2009) C. G. Crist et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- NF-κB–YY1–miR-29 Regulatory Circuitry in Skeletal Myogenesis and Rhabdomyosarcoma
- (2008) Huating Wang et al. CANCER CELL
- microRNA-133a regulates cardiomyocyte proliferation and suppresses smooth muscle gene expression in the heart
- (2008) N. Liu et al. GENES & DEVELOPMENT
- All muscle satellite cells are equal, but are some more equal than others?
- (2008) P. S. Zammit JOURNAL OF CELL SCIENCE
- Role of retrotransposon-derived imprinted gene, Rtl1, in the feto-maternal interface of mouse placenta
- (2008) Yoichi Sekita et al. NATURE GENETICS
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