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Cancers with increasing incidence trends in the United States: 1999 through 2008

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Oncogenic Kras Maintains Pancreatic Tumors through Regulation of Anabolic Glucose Metabolism

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Glucose-Independent Glutamine Metabolism via TCA Cycling for Proliferation and Survival in B Cells

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MUC1 mucin stabilizes and activates hypoxia-inducible factor 1 alpha to regulate metabolism in pancreatic cancer

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Phosphofructokinase 1 Glycosylation Regulates Cell Growth and Metabolism

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The hexosamine biosynthetic pathway and O-GlcNAcylation drive the expression of β-catenin and cell proliferation

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Proteomic Analysis Reveals Warburg Effect and Anomalous Metabolism of Glutamine in Pancreatic Cancer Cells

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Oncogenic K-Ras decouples glucose and glutamine metabolism to support cancer cell growth

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Hypoxia promotes isocitrate dehydrogenase-dependent carboxylation of  -ketoglutarate to citrate to support cell growth and viability

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Metabolism strikes back: metabolic flux regulates cell signaling

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Stromal biology and therapy in pancreatic cancer

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Tumour hypoxia induces a metabolic shift causing acidosis: a common feature in cancer

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Radiosensitization and Stromal Imaging Response Correlates for the HIF-1 Inhibitor PX-478 Given with or without Chemotherapy in Pancreatic Cancer

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O-GlcNAc signaling: a metabolic link between diabetes and cancer?

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Tumor metabolism of lactate: the influence and therapeutic potential for MCT and CD147 regulation

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CD147 silencing inhibits lactate transport and reduces malignant potential of pancreatic cancer cells in in vivo and in vitro models

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Glucose Deprivation Contributes to the Development of KRAS Pathway Mutations in Tumor Cells

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Redox mechanisms switch on hypoxia-dependent epithelial–mesenchymal transition in cancer cells

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