Helicobacter pylori -glutamyl transpeptidase and vacuolating cytotoxin promote gastric persistence and immune tolerance
出版年份 2013 全文链接
标题
Helicobacter pylori -glutamyl transpeptidase and vacuolating cytotoxin promote gastric persistence and immune tolerance
作者
关键词
-
出版物
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 110, Issue 8, Pages 3047-3052
出版商
Proceedings of the National Academy of Sciences
发表日期
2013-02-05
DOI
10.1073/pnas.1211248110
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Vacuolating Cytotoxin and Variants in Atg16L1 That Disrupt Autophagy Promote Helicobacter pylori Infection in Humans
- (2012) Deepa Raju et al. GASTROENTEROLOGY
- DC-derived IL-18 drives Treg differentiation, murine Helicobacter pylori–specific immune tolerance, and asthma protection
- (2012) Mathias Oertli et al. JOURNAL OF CLINICAL INVESTIGATION
- Intoxication strategy of Helicobacter pylori VacA toxin
- (2012) Patrice Boquet et al. TRENDS IN MICROBIOLOGY
- H. pylori exploits and manipulates innate and adaptive immune cell signaling pathways to establish persistent infection
- (2011) Anne Müller et al. Cell Communication and Signaling
- Stimulation of dendritic cells with Helicobacter pylori vacuolating cytotoxin negatively regulates their maturation via the restoration of E2F1
- (2011) J. M. Kim et al. CLINICAL AND EXPERIMENTAL IMMUNOLOGY
- Dendritic Cells Prevent Rather Than Promote Immunity Conferred by a Helicobacter Vaccine Using a Mycobacterial Adjuvant
- (2011) Iris Hitzler et al. GASTROENTEROLOGY
- Helicobacter pylori DNA decreases pro-inflammatory cytokine production by dendritic cells and attenuates dextran sodium sulphate-induced colitis
- (2011) J. Luther et al. GUT
- Helicobacter pylori infection prevents allergic asthma in mouse models through the induction of regulatory T cells
- (2011) Isabelle C. Arnold et al. JOURNAL OF CLINICAL INVESTIGATION
- Tolerance Rather Than Immunity Protects From Helicobacter pylori–Induced Gastric Preneoplasia
- (2010) Isabelle C. Arnold et al. GASTROENTEROLOGY
- Helicobacter pylori Induces miR-155 in T Cells in a cAMP-Foxp3-Dependent Manner
- (2010) Lina Fassi Fehri et al. PLoS One
- Helicobacter pylori Impairs Murine Dendritic Cell Responses to Infection
- (2010) Ya-Hui Wang et al. PLoS One
- Microevolution of Helicobacter pylori during Prolonged Infection of Single Hosts and within Families
- (2010) Giovanna Morelli et al. PLoS Genetics
- Helicobacter pylori Immune Escape Is Mediated by Dendritic Cell–Induced Treg Skewing and Th17 Suppression in Mice
- (2009) John Y. Kao et al. GASTROENTEROLOGY
- Association between Helicobacter pylori infection and inflammatory bowel disease
- (2009) Jay Luther et al. INFLAMMATORY BOWEL DISEASES
- The CD4+ T Cell-Mediated IFN- Response to Helicobacter Infection Is Essential for Clearance and Determines Gastric Cancer Risk
- (2009) A. Sayi et al. JOURNAL OF IMMUNOLOGY
- What are the consequences of the disappearing human microbiota?
- (2009) Martin J. Blaser et al. NATURE REVIEWS MICROBIOLOGY
- Acquisition of Helicobacter Pylori Infection in Early Childhood: Independent Contributions of Infected Mothers, Fathers and Siblings
- (2008) Maria Weyermann et al. AMERICAN JOURNAL OF GASTROENTEROLOGY
- Regulation of Gastric Carcinogenesis by Helicobacter pylori Virulence Factors
- (2008) A. T. Franco et al. CANCER RESEARCH
- Integrin Subunit CD18 Is the T-Lymphocyte Receptor for the Helicobacter pylori Vacuolating Cytotoxin
- (2008) Xaver Sewald et al. Cell Host & Microbe
- Helicobacter pylori Gastritis in Children Is Associated With a Regulatory T-Cell Response
- (2008) Paul R. Harris et al. GASTROENTEROLOGY
- Helicobacter pylori-induced peptic ulcer disease is associated with inadequate regulatory T cell responses
- (2008) K Robinson et al. GUT
- Helicobacter pyloriColonization Is Inversely Associated with Childhood Asthma
- (2008) Yu Chen et al. JOURNAL OF INFECTIOUS DISEASES
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started