AMP-activated protein kinase (AMPK) activity is not required for neuronal development but regulates axogenesis during metabolic stress
出版年份 2011 全文链接
标题
AMP-activated protein kinase (AMPK) activity is not required for neuronal development but regulates axogenesis during metabolic stress
作者
关键词
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出版物
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 108, Issue 14, Pages 5849-5854
出版商
Proceedings of the National Academy of Sciences
发表日期
2011-03-22
DOI
10.1073/pnas.1013660108
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Metformin, Independent of AMPK, Inhibits mTORC1 in a Rag GTPase-Dependent Manner
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- AMP-Activated Protein Kinase Phosphorylates Retinoblastoma Protein to Control Mammalian Brain Development
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- KCC2 Expression Promotes the Termination of Cortical Interneuron Migration in a Voltage-Sensitive Calcium-Dependent Manner
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- LKB1 regulates polarity remodeling and adherens junction formation in the Drosophila eye
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- Hypothalamic CaMKK2 Contributes to the Regulation of Energy Balance
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- Tuberous sclerosis complex proteins control axon formation
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- AMPK Phosphorylation of Raptor Mediates a Metabolic Checkpoint
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- Caenorhabditis elegans dauers need LKB1/AMPK to ration lipid reserves and ensure long-term survival
- (2008) Patrick Narbonne et al. NATURE
- LKB1 and AMPK in cell polarity and division
- (2008) Tyisha Williams et al. TRENDS IN CELL BIOLOGY
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