HACD1, a regulator of membrane composition and fluidity, promotes myoblast fusion and skeletal muscle growth

Phospholipase D1 facilitates second-phase myoblast fusion and skeletal muscle regeneration

(2015) Shuzhi Teng et al.   MOLECULAR BIOLOGY OF THE CELL

Pathophysiological concepts in the congenital myopathies: blurring the boundaries, sharpening the focus

(2015) Gianina Ravenscroft et al.   BRAIN

Inhibition of very long acyl chain sphingolipid synthesis modifies membrane dynamics during plant cytokinesis

(2014) Diana Molino et al.   BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR AND CELL BIOLOGY OF LIPIDS

GRAF1 promotes ferlin-dependent myoblast fusion

(2014) Kaitlin C. Lenhart et al.   DEVELOPMENTAL BIOLOGY

Myomaker is essential for muscle regeneration

(2014) Douglas P. Millay et al.   GENES & DEVELOPMENT

Lysophosphatidylcholine Reversibly Arrests Pore Expansion during Syncytium Formation Mediated by Diverse Viral Fusogens

(2014) M. Ciechonska et al.   JOURNAL OF VIROLOGY

Congenital myopathy is caused by mutation of HACD1

(2013) Emad Muhammad et al.   HUMAN MOLECULAR GENETICS

Extracellular annexins and dynamin are important for sequential steps in myoblast fusion

(2013) Evgenia Leikina et al.   JOURNAL OF CELL BIOLOGY

Phosphatidylserine receptor BAI1 and apoptotic cells as new promoters of myoblast fusion

(2013) Amelia E. Hochreiter-Hufford et al.   NATURE

Myomaker is a membrane activator of myoblast fusion and muscle formation

(2013) Douglas P. Millay et al.   NATURE

Genetic basis of cell–cell fusion mechanisms

(2013) Pablo S. Aguilar et al.   TRENDS IN GENETICS

Controlling cancer through the autotaxin–lysophosphatidic acid receptor axis

(2012) Mari Gotoh et al.   BIOCHEMICAL SOCIETY TRANSACTIONS

Myogenic differentiation and lipid-raft composition of L6 skeletal muscle cells are modulated by PUFAs

(2012) Anne Briolay et al.   BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES

Congenital Myopathies: An Update

(2012) Jessica R. Nance et al.   Current Neurology and Neuroscience Reports

Myoblast fusion: lessons from flies and mice

(2012) S. M. Abmayr et al.   DEVELOPMENT

Very long-chain fatty acids: elongation, physiology and related disorders

(2012) A. Kihara   JOURNAL OF BIOCHEMISTRY

Centronuclear Myopathy in Labrador Retrievers: A Recent Founder Mutation in the PTPLA Gene Has Rapidly Disseminated Worldwide

(2012) Marie Maurer et al.   PLoS One

Building Muscle: Molecular Regulation of Myogenesis

(2012) C. F. Bentzinger et al.   Cold Spring Harbor Perspectives in Biology

Protein Tyrosine Phosphatase-Like A Regulates Myoblast Proliferation and Differentiation through MyoG and the Cell Cycling Signaling Pathway

(2011) X. Lin et al.   MOLECULAR AND CELLULAR BIOLOGY

Centronuclear myopathies: A widening concept

(2010) Norma Beatriz Romero   NEUROMUSCULAR DISORDERS

ELOVL1 production of C24 acyl-CoAs is linked to C24 sphingolipid synthesis

(2010) Y. Ohno et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA

Metabolism and atherogenic disease association of lysophosphatidylcholine

(2009) Gerd Schmitz et al.   ATHEROSCLEROSIS

Modulation of proliferation and differentiation of C2C12 skeletal muscle cells by fatty acids

(2009) Ju-Hye Lee et al.   LIFE SCIENCES

The key roles of elongases and desaturases in mammalian fatty acid metabolism: Insights from transgenic mice

(2009) Hervé Guillou et al.   PROGRESS IN LIPID RESEARCH

Characterization of four mammalian 3-hydroxyacyl-CoA dehydratases involved in very long-chain fatty acid synthesis

(2008) Mika Ikeda et al.   FEBS LETTERS

Membrane Topology and Essential Amino Acid Residues of Phs1, a 3-Hydroxyacyl-CoA Dehydratase Involved in Very Long-chain Fatty Acid Elongation

(2008) Akio Kihara et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

What’s new in congenital myopathies?

(2008) Kathryn North   NEUROMUSCULAR DISORDERS

Centronuclear (myotubular) myopathy

(2008) Heinz Jungbluth et al.   Orphanet Journal of Rare Diseases

The atypical Rac activator Dock180 (Dock1) regulates myoblast fusion in vivo

(2008) M. Laurin et al.   PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA