Expression of Nitric Oxide-Transporting Aquaporin-1 Is Controlled by KLF2 and Marks Non-Activated Endothelium In Vivo

Adipose tissue-derived stromal cells acquire endothelial-like features upon reprogramming with SOX18

(2014) R.D. Fontijn et al.   Stem Cell Research

Cardiac aquaporins

(2013) Arkady Rutkovskiy et al.   BASIC RESEARCH IN CARDIOLOGY

Genetic deletion of aquaporin-1 results in microcardia and low blood pressure in mouse with intact nitric oxide-dependent relaxation, but enhanced prostanoids-dependent relaxation

(2013) V. Montiel et al.   PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY

Laminar shear stress induces the expression of aquaporin 1 in endothelial cells involved in wound healing

(2012) Gyeong In Mun et al.   BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS

Atheroprotective communication between endothelial cells and smooth muscle cells through miRNAs

(2012) Eduard Hergenreider et al.   NATURE CELL BIOLOGY

KLF2-induced actin shear fibers control both alignment to flow and JNK signaling in vascular endothelium

(2009) R. A. Boon et al.   BLOOD

Aquaporin 1 is required for hypoxia-inducible angiogenesis in human retinal vascular endothelial cells

(2008) Kentaro Kaneko et al.   MICROVASCULAR RESEARCH

SOX-18 controls endothelial-specific claudin-5 gene expression and barrier function

(2007) Ruud D. Fontijn et al.   AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY