标题
Upregulation of TMEM16A Protein in Bronchial Epithelial Cells by Bacterial Pyocyanin
作者
关键词
-
出版物
PLoS One
Volume 10, Issue 6, Pages e0131775
出版商
Public Library of Science (PLoS)
发表日期
2015-06-30
DOI
10.1371/journal.pone.0131775
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Characteristics and Pharmacological Regulation of Epithelial Na+ Channel (ENaC) and Epithelial Na+ Transport
- (2014) Yoshinori Marunaka JOURNAL OF PHARMACOLOGICAL SCIENCES
- Impaired mucus detachment disrupts mucociliary transport in a piglet model of cystic fibrosis
- (2014) M. J. Hoegger et al. SCIENCE
- Bicarbonate and functional CFTR channel are required for proper mucin secretion and link cystic fibrosis with its mucus phenotype
- (2012) Jenny K. Gustafsson et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Association of TMEM16A chloride channel overexpression with airway goblet cell metaplasia
- (2012) Paolo Scudieri et al. JOURNAL OF PHYSIOLOGY-LONDON
- Reduced airway surface pH impairs bacterial killing in the porcine cystic fibrosis lung
- (2012) Alejandro A. Pezzulo et al. NATURE
- Dynamic modulation of ANO1/TMEM16A HCO3- permeability by Ca2+/calmodulin
- (2012) J. Jung et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction
- (2012) F. Huang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Pseudomonas aeruginosa pyocyanin causes airway goblet cell hyperplasia and metaplasia and mucus hypersecretion by inactivating the transcriptional factor FoxA2
- (2011) Yonghua Hao et al. CELLULAR MICROBIOLOGY
- Interleukin-13–Induced Mucous Metaplasia Increases Susceptibility of Human Airway Epithelium to Rhinovirus Infection
- (2010) Marrah E. Lachowicz-Scroggins et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Goblet Cells Are Derived from aFOXJ1-Expressing Progenitor in a Human Airway Epithelium
- (2010) Jonathan Turner et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Coupled Nucleotide and Mucin Hypersecretion from Goblet-Cell Metaplastic Human Airway Epithelium
- (2010) Seiko F. Okada et al. AMERICAN JOURNAL OF RESPIRATORY CELL AND MOLECULAR BIOLOGY
- Allergic airway inflammation induces a pro-secretory epithelial ion transport phenotype in mice
- (2010) P. Anagnostopoulou et al. EUROPEAN RESPIRATORY JOURNAL
- Pseudomonas aeruginosa Exotoxin Pyocyanin Causes Cystic Fibrosis Airway Pathogenesis
- (2009) Charles C. Caldwell et al. AMERICAN JOURNAL OF PATHOLOGY
- Loss of TMEM16A Causes a Defect in Epithelial Ca2+-dependent Chloride Transport
- (2009) Jiraporn Ousingsawat et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Upregulation ofMUC5ACgene expression by IL-4 through CREB in Human airway epithelial cells
- (2009) Cheol Hong Kim et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Substance P stimulates human airway submucosal gland secretion mainly via a CFTR-dependent process
- (2009) Jae Young Choi et al. JOURNAL OF CLINICAL INVESTIGATION
- Normal mouse intestinal mucus release requires cystic fibrosis transmembrane regulator–dependent bicarbonate secretion
- (2009) Mary Abigail S. Garcia et al. JOURNAL OF CLINICAL INVESTIGATION
- CFTR Function and Prospects for Therapy
- (2008) John R. Riordan Annual Review of Biochemistry
- Expression Cloning of TMEM16A as a Calcium-Activated Chloride Channel Subunit
- (2008) Björn Christian Schroeder et al. CELL
- Oxidative stress caused by pyocyanin impairs CFTR Cl− transport in human bronchial epithelial cells
- (2008) Christian Schwarzer et al. FREE RADICAL BIOLOGY AND MEDICINE
- TMEM16A confers receptor-activated calcium-dependent chloride conductance
- (2008) Young Duk Yang et al. NATURE
- TMEM16A, A Membrane Protein Associated with Calcium-Dependent Chloride Channel Activity
- (2008) A. Caputo et al. SCIENCE
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