Transcription Factors OVOL1 and OVOL2 Induce the Mesenchymal to Epithelial Transition in Human Cancer

The Cancer Cell Line Encyclopedia enables predictive modelling of anticancer drug sensitivity

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Differential gene and transcript expression analysis of RNA-seq experiments with TopHat and Cufflinks

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Complex changes in alternative pre-mRNA splicing play a central role in the epithelial-to-mesenchymal transition (EMT)

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Paracrine and Autocrine Signals Induce and Maintain Mesenchymal and Stem Cell States in the Breast

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CD44 splice isoform switching in human and mouse epithelium is essential for epithelial-mesenchymal transition and breast cancer progression

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Human prostate cancer metastases target the hematopoietic stem cell niche to establish footholds in mouse bone marrow

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TGF-β drives epithelial-mesenchymal transition through δEF1-mediated downregulation of ESRP

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A Perspective on Cancer Cell Metastasis

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An EMT–Driven Alternative Splicing Program Occurs in Human Breast Cancer and Modulates Cellular Phenotype

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An ESRP-regulated splicing programme is abrogated during the epithelial–mesenchymal transition

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Alternative pre-mRNA splicing regulation in cancer: pathways and programs unhinged

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IL-4 induces cathepsin protease activity in tumor-associated macrophages to promote cancer growth and invasion

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ConceptGen: a gene set enrichment and gene set relation mapping tool

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TopHat: discovering splice junctions with RNA-Seq

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CD4+ T Cells Regulate Pulmonary Metastasis of Mammary Carcinomas by Enhancing Protumor Properties of Macrophages

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Epithelial-Mesenchymal Transitions in Development and Disease

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Ovol2 Suppresses Cell Cycling and Terminal Differentiation of Keratinocytes by Directly Repressing c-MycandNotch1

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CCL2 and Interleukin-6 Promote Survival of Human CD11b+Peripheral Blood Mononuclear Cells and Induce M2-type Macrophage Polarization

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ESRP1 and ESRP2 Are Epithelial Cell-Type-Specific Regulators of FGFR2 Splicing

(2009) Claude C. Warzecha et al.   MOLECULAR CELL

Epithelial-Mesenchymal Transition: At the Crossroads of Development and Tumor Metastasis

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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

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The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

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EPB41L5 functions to post-transcriptionally regulate cadherin and integrin during epithelial–mesenchymal transition

(2008) Mariko Hirano et al.   JOURNAL OF CELL BIOLOGY

The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1

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