标题
Role of Hepcidin in the Setting of Hypoferremia during Acute Inflammation
作者
关键词
-
出版物
PLoS One
Volume 8, Issue 4, Pages e61050
出版商
Public Library of Science (PLoS)
发表日期
2014-04-30
DOI
10.1371/journal.pone.0061050
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Toll-Like Receptor Signal Adaptor Protein MyD88 Is Required for Sustained Endotoxin-Induced Acute Hypoferremic Response in Mice
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- Toll-like receptor signalling in liver disease: ER stress the missing link?
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- Lipocalin 2 Deficiency Dysregulates Iron Homeostasis and Exacerbates Endotoxin-Induced Sepsis
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- Low hepcidin accounts for the proinflammatory status associated with iron deficiency
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- A Ferroportin Transcript that Lacks an Iron-Responsive Element Enables Duodenal and Erythroid Precursor Cells to Evade Translational Repression
- (2009) De-Liang Zhang et al. Cell Metabolism
- Hepcidin targets ferroportin for degradation in hepatocytes
- (2009) G. Ramey et al. HAEMATOLOGICA
- Lack of the bone morphogenetic protein BMP6 induces massive iron overload
- (2009) Delphine Meynard et al. NATURE GENETICS
- Iron metabolism in the anemia of chronic disease
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- Iron regulates phosphorylation of Smad1/5/8 and gene expression of Bmp6, Smad7, Id1, and Atoh8 in the mouse liver
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- LPS/TLR4 signal transduction pathway
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