Lipoteichoic Acid from Staphylococcus aureus Induces Lung Endothelial Cell Barrier Dysfunction: Role of Reactive Oxygen and Nitrogen Species

Cooperation of TLR2 with MyD88, PI3K, and Rac1 in Lipoteichoic Acid–Induced cPLA2/COX-2–Dependent Airway Inflammatory Responses

(2010) I-Ta Lee et al.   AMERICAN JOURNAL OF PATHOLOGY

Natural Staphylococcus aureus-derived peptidoglycan fragments activate NOD2 and act as potent costimulators of the innate immune system exclusively in the presence of TLR signals

(2010) Thomas Volz et al.   FASEB JOURNAL

IRAK1 and IRAK4 Promote Phosphorylation, Ubiquitination, and Degradation of MyD88 Adaptor-like (Mal)

(2010) Aisling Dunne et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

IRAK-1 Contributes to Lipopolysaccharide-induced Reactive Oxygen Species Generation in Macrophages by Inducing NOX-1 Transcription and Rac1 Activation and Suppressing the Expression of Antioxidative Enzymes

(2009) Urmila Maitra et al.   JOURNAL OF BIOLOGICAL CHEMISTRY

Reduction of biofilm formation with trisodium citrate in haemodialysis catheters: a randomized controlled trial

(2009) J. W. Bosma et al.   NEPHROLOGY DIALYSIS TRANSPLANTATION

Lung Inflammation Induced by Lipoteichoic Acid or Lipopolysaccharide in Humans

(2008) Jacobien J. Hoogerwerf et al.   AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE