Antagonism of miR-21 Reverses Epithelial-Mesenchymal Transition and Cancer Stem Cell Phenotype through AKT/ERK1/2 Inactivation by Targeting PTEN
出版年份 2012 全文链接
标题
Antagonism of miR-21 Reverses Epithelial-Mesenchymal Transition and Cancer Stem Cell Phenotype through AKT/ERK1/2 Inactivation by Targeting PTEN
作者
关键词
-
出版物
PLoS One
Volume 7, Issue 6, Pages e39520
出版商
Public Library of Science (PLoS)
发表日期
2012-06-27
DOI
10.1371/journal.pone.0039520
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Activation of β-catenin and Akt pathways by Twist are critical for the maintenance of EMT associated cancer stem cell-like characters
- (2011) Junlin Li et al. BMC CANCER
- RETRACTED: Notch-1 induces epithelial–mesenchymal transition consistent with cancer stem cell phenotype in pancreatic cancer cells
- (2011) Bin Bao et al. CANCER LETTERS
- MicroRNA Sequence and Expression Analysis in Breast Tumors by Deep Sequencing
- (2011) T. A. Farazi et al. CANCER RESEARCH
- Cisplatin treatment of primary and metastatic epithelial ovarian carcinomas generates residual cells with mesenchymal stem cell-like profile
- (2011) Ardian Latifi et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Re-expression of miR-21 contributes to migration and invasion by inducing epithelial-mesenchymal transition consistent with cancer stem cell characteristics in MCF-7 cells
- (2011) Mingli Han et al. MOLECULAR AND CELLULAR BIOCHEMISTRY
- Anti-Tumor Activity of a Novel Compound-CDF Is Mediated by Regulating miR-21, miR-200, and PTEN in Pancreatic Cancer
- (2011) Bin Bao et al. PLoS One
- Epithelial to Mesenchymal Transition by TGFβ-1 Induction Increases Stemness Characteristics in Primary Non Small Cell Lung Cancer Cell Line
- (2011) Giuseppe Pirozzi et al. PLoS One
- Endothelial Induced EMT in Breast Epithelial Cells with Stem Cell Properties
- (2011) Valgardur Sigurdsson et al. PLoS One
- Activation of Akt and MAPK pathways enhances the tumorigenicity of CD133+ primary colon cancer cells
- (2010) Y. K. Wang et al. CARCINOGENESIS
- Epithelial Mesenchymal Transition Traits in Human Breast Cancer Cell Lines Parallel the CD44hi/CD24lo/- Stem Cell Phenotype in Human Breast Cancer
- (2010) Tony Blick et al. JOURNAL OF MAMMARY GLAND BIOLOGY AND NEOPLASIA
- Bmi1 is essential in Twist1-induced epithelial–mesenchymal transition
- (2010) Muh-Hwa Yang et al. NATURE CELL BIOLOGY
- Downregulation of microRNAs directs the EMT and invasive potential of anaplastic thyroid carcinomas
- (2010) J Braun et al. ONCOGENE
- Nuclear Factor-κB Enhances ErbB2-Induced Mammary Tumorigenesis and Neoangiogenesis in Vivo
- (2009) Manran Liu et al. AMERICAN JOURNAL OF PATHOLOGY
- MicroRNA dynamics in the stages of tumorigenesis correlate with hallmark capabilities of cancer
- (2009) P. Olson et al. GENES & DEVELOPMENT
- p21CIP1attenuates Ras- and c-Myc-dependent breast tumor epithelial mesenchymal transition and cancer stem cell-like gene expression in vivo
- (2009) Manran Liu et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- MicroRNAs Differentially Regulated by Akt Isoforms Control EMT and Stem Cell Renewal in Cancer Cells
- (2009) D. Iliopoulos et al. Science Signaling
- Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype
- (2008) D. Sarrio et al. CANCER RESEARCH
- The Epithelial-Mesenchymal Transition Generates Cells with Properties of Stem Cells
- (2008) Sendurai A. Mani et al. CELL
- MicroRNA-21 targets tumor suppressor genes in invasion and metastasis
- (2008) Shuomin Zhu et al. CELL RESEARCH
- MicroRNA 21 Promotes Glioma Invasion by Targeting Matrix Metalloproteinase Regulators
- (2008) G. Gabriely et al. MOLECULAR AND CELLULAR BIOLOGY
- Generation of Breast Cancer Stem Cells through Epithelial-Mesenchymal Transition
- (2008) Anne-Pierre Morel et al. PLoS One
Add your recorded webinar
Do you already have a recorded webinar? Grow your audience and get more views by easily listing your recording on Peeref.
Upload NowBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started