标题
The ζ Toxin Induces a Set of Protective Responses and Dormancy
作者
关键词
-
出版物
PLoS One
Volume 7, Issue 1, Pages e30282
出版商
Public Library of Science (PLoS)
发表日期
2012-01-26
DOI
10.1371/journal.pone.0030282
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Toxins-antitoxins: diversity, evolution and function
- (2011) Finbarr Hayes et al. CRITICAL REVIEWS IN BIOCHEMISTRY AND MOLECULAR BIOLOGY
- Balancing at survival's edge: the structure and adaptive benefits of prokaryotic toxin–antitoxin partners
- (2011) Tim R Blower et al. CURRENT OPINION IN STRUCTURAL BIOLOGY
- The Escherichia coli Extracellular Death Factor EDF Induces the Endoribonucleolytic Activities of the Toxins MazF and ChpBK
- (2011) Maria Belitsky et al. MOLECULAR CELL
- Regulation of growth and death in Escherichia coli by toxin–antitoxin systems
- (2011) Yoshihiro Yamaguchi et al. NATURE REVIEWS MICROBIOLOGY
- Diversity of bacterial type II toxin–antitoxin systems: a comprehensive search and functional analysis of novel families
- (2011) Raphaël Leplae et al. NUCLEIC ACIDS RESEARCH
- A Novel Mechanism of Programmed Cell Death in Bacteria by Toxin–Antitoxin Systems Corrupts Peptidoglycan Synthesis
- (2011) Hannes Mutschler et al. PLOS BIOLOGY
- Toxin–antitoxin systems: why so many, what for?
- (2010) Laurence Van Melderen CURRENT OPINION IN MICROBIOLOGY
- Differential Responses of Bacillus subtilis rRNA Promoters to Nutritional Stress
- (2010) W. Samarrai et al. JOURNAL OF BACTERIOLOGY
- YeeV is an Escherichia coli toxin that inhibits cell division by targeting the cytoskeleton proteins, FtsZ and MreB
- (2010) Qian Tan et al. MOLECULAR MICROBIOLOGY
- The relBE2Spn Toxin-Antitoxin System of Streptococcus pneumoniae: Role in Antibiotic Tolerance and Functional Conservation in Clinical Isolates
- (2010) Concha Nieto et al. PLoS One
- Ciprofloxacin Causes Persister Formation by Inducing the TisB toxin in Escherichia coli
- (2010) Tobias Dörr et al. PLOS BIOLOGY
- PCR-based plasmid typing inEnterococcus faeciumstrains reveals widely distributed pRE25-, pRUM-, pIP501- and pHTβ-related replicons associated with glycopeptide resistance and stabilizing toxin–antitoxin systems
- (2009) Torill C.S. Rosvoll et al. FEMS IMMUNOLOGY AND MEDICAL MICROBIOLOGY
- Overexpression of MazFSa in Staphylococcus aureus Induces Bacteriostasis by Selectively Targeting mRNAs for Cleavage
- (2009) Z. Fu et al. JOURNAL OF BACTERIOLOGY
- Rejuvenation of CcdB-Poisoned Gyrase by an Intrinsically Disordered Protein Domain
- (2009) Natalie De Jonge et al. MOLECULAR CELL
- Molecular Mechanisms of HipA-Mediated Multidrug Tolerance and Its Neutralization by HipB
- (2009) M. A. Schumacher et al. SCIENCE
- Bacterial Toxin–Antitoxin Systems: More Than Selfish Entities?
- (2009) Laurence Van Melderen et al. PLoS Genetics
- SOS Response Induces Persistence to Fluoroquinolones in Escherichia coli
- (2009) Tobias Dörr et al. PLoS Genetics
- (p)ppGpp: Still Magical?
- (2008) Katarzyna Potrykus et al. Annual Review of Microbiology
- MazF, an mRNA Interferase, Mediates Programmed Cell Death during Multicellular Myxococcus Development
- (2008) Hirofumi Nariya et al. CELL
- Control of bacterial transcription, translation and replication by (p)ppGpp
- (2008) Anjana Srivatsan et al. CURRENT OPINION IN MICROBIOLOGY
- Crystal Structure ofEscherichia coliMazG, the Regulator of Nutritional Stress Response
- (2008) Sujin Lee et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Structure and Proposed Activity of a Member of the VapBC Family of Toxin-Antitoxin Systems
- (2008) Linda Miallau et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- A small SOS-induced toxin is targeted against the inner membrane in Escherichia coli
- (2008) Cecilia Unoson et al. MOLECULAR MICROBIOLOGY
- The Communication Factor EDF and the Toxin–Antitoxin Module mazEF Determine the Mode of Action of Antibiotics
- (2008) Ilana Kolodkin-Gal et al. PLOS BIOLOGY
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