Type I IFN Promotes IL-10 Production from T Cells to Suppress Th17 Cells and Th17-Associated Autoimmune Inflammation
出版年份 2011 全文链接
标题
Type I IFN Promotes IL-10 Production from T Cells to Suppress Th17 Cells and Th17-Associated Autoimmune Inflammation
作者
关键词
-
出版物
PLoS One
Volume 6, Issue 12, Pages e28432
出版商
Public Library of Science (PLoS)
发表日期
2011-12-07
DOI
10.1371/journal.pone.0028432
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Cutting Edge: IL-27 Is a Potent Inducer of IL-10 but Not FoxP3 in Murine T Cells
- (2014) M. Batten et al. JOURNAL OF IMMUNOLOGY
- Beta-interferons in multiple sclerosis
- (2010) Sourabh Aggarwal et al. Annals of Indian Academy of Neurology
- Role of Th17 cells in human autoimmune arthritis
- (2010) Jan Leipe et al. ARTHRITIS AND RHEUMATISM
- Th17 and Regulatory T Cells in Mediating and Restraining Inflammation
- (2010) Dan R. Littman et al. CELL
- Interferon beta inhibits the Th17 cell-mediated autoimmune response in patients with relapsing–remitting multiple sclerosis
- (2010) Xin Zhang et al. CLINICAL NEUROLOGY AND NEUROSURGERY
- Functional and Pathogenic Differences of Th1 and Th17 Cells in Experimental Autoimmune Encephalomyelitis
- (2010) Helena S. Domingues et al. PLoS One
- New lessons about old molecules: how type I interferons shape Th1/Th17-mediated autoimmunity in the CNS
- (2010) Marco Prinz et al. TRENDS IN MOLECULAR MEDICINE
- Regulation of autoimmune encephalomyelitis by toll-like receptors
- (2009) Monica Marta et al. AUTOIMMUNITY REVIEWS
- RORC2: The master of human Th17 cell programming
- (2009) Derya Unutmaz EUROPEAN JOURNAL OF IMMUNOLOGY
- Global Mapping of H3K4me3 and H3K27me3 Reveals Specificity and Plasticity in Lineage Fate Determination of Differentiating CD4+ T Cells
- (2009) Gang Wei et al. IMMUNITY
- Late Developmental Plasticity in the T Helper 17 Lineage
- (2009) Yun Kyung Lee et al. IMMUNITY
- Th1, Th17, and Th9 Effector Cells Induce Experimental Autoimmune Encephalomyelitis with Different Pathological Phenotypes
- (2009) A. Jager et al. JOURNAL OF IMMUNOLOGY
- IL-27 Is a Key Regulator of IL-10 and IL-17 Production by Human CD4+ T Cells
- (2009) G. Murugaiyan et al. JOURNAL OF IMMUNOLOGY
- CD39+Foxp3+ Regulatory T Cells Suppress Pathogenic Th17 Cells and Are Impaired in Multiple Sclerosis
- (2009) J. M. Fletcher et al. JOURNAL OF IMMUNOLOGY
- A protective function for interleukin 17A in T cell–mediated intestinal inflammation
- (2009) William O'Connor Jr et al. NATURE IMMUNOLOGY
- CD4+ Regulatory T Cells Control TH17 Responses in a Stat3-Dependent Manner
- (2009) A. Chaudhry et al. SCIENCE
- Engagement of the Type I Interferon Receptor on Dendritic Cells Inhibits T Helper 17 Cell Development: Role of Intracellular Osteopontin
- (2008) Mari L. Shinohara et al. IMMUNITY
- Regulatory T Cell-Derived Interleukin-10 Limits Inflammation at Environmental Interfaces
- (2008) Yuri P. Rubtsov et al. IMMUNITY
- Distinct and Nonredundant In Vivo Functions of IFNAR on Myeloid Cells Limit Autoimmunity in the Central Nervous System
- (2008) Marco Prinz et al. IMMUNITY
- The Biological Functions of T Helper 17 Cell Effector Cytokines in Inflammation
- (2008) Wenjun Ouyang et al. IMMUNITY
- Molecular Antagonism and Plasticity of Regulatory and Inflammatory T Cell Programs
- (2008) Xuexian O. Yang et al. IMMUNITY
- The type I IFN induction pathway constrains Th17-mediated autoimmune inflammation in mice
- (2008) Beichu Guo et al. JOURNAL OF CLINICAL INVESTIGATION
- Interleukin-17 in transverse myelitis and multiple sclerosis
- (2008) J GRABER et al. JOURNAL OF NEUROIMMUNOLOGY
- A shift from adaptive to innate immunity: a potential mechanism of disease progression in multiple sclerosis
- (2008) Howard L. Weiner JOURNAL OF NEUROLOGY
- TGF-β-induced Foxp3 inhibits TH17 cell differentiation by antagonizing RORγt function
- (2008) Liang Zhou et al. NATURE
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