The c-Jun N-terminal kinase 1 (JNK1) in spinal astrocytes is required for the maintenance of bilateral mechanical allodynia under a persistent inflammatory pain condition

Glycine Inhibitory Dysfunction Induces a Selectively Dynamic, Morphine-Resistant, and Neurokinin 1 Receptor- Independent Mechanical Allodynia

(2009) L. S. Miraucourt et al.   JOURNAL OF NEUROSCIENCE

JNK-Induced MCP-1 Production in Spinal Cord Astrocytes Contributes to Central Sensitization and Neuropathic Pain

(2009) Y.-J. Gao et al.   JOURNAL OF NEUROSCIENCE

MAP kinase and pain

(2008) Ru-Rong Ji et al.   BRAIN RESEARCH REVIEWS

Transforming growth factor-activated kinase 1 induced in spinal astrocytes contributes to mechanical hypersensitivity after nerve injury

(2008) Hirokazu Katsura et al.   GLIA

Activation of the Neuronal Extracellular Signal-Regulated Kinase 2 in the Spinal Cord Dorsal Horn Is Required for Complete Freund's Adjuvant-Induced Pain Hypersensitivity

(2008) Q. Xu et al.   JOURNAL OF NEUROSCIENCE

Innocuous, Not Noxious, Input Activates PKC  Interneurons of the Spinal Dorsal Horn via Myelinated Afferent Fibers

(2008) S. Neumann et al.   JOURNAL OF NEUROSCIENCE

Pathological and protective roles of glia in chronic pain

(2008) Erin D. Milligan et al.   NATURE REVIEWS NEUROSCIENCE

Roles of peripheral mitogen-activated protein kinases in melittin-induced nociception and hyperalgesia

(2008) J. Hao et al.   NEUROSCIENCE

Activation of JNK pathway in persistent pain

(2008) Yong-Jing Gao et al.   NEUROSCIENCE LETTERS