Gene amplification of the histone methyltransferase SETDB1 contributes to human lung tumorigenesis
出版年份 2013 全文链接
标题
Gene amplification of the histone methyltransferase SETDB1 contributes to human lung tumorigenesis
作者
关键词
-
出版物
ONCOGENE
Volume 33, Issue 21, Pages 2807-2813
出版商
Springer Nature
发表日期
2013-06-17
DOI
10.1038/onc.2013.239
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- The Cancer Cell Line Encyclopedia enables predictive modelling of anticancer drug sensitivity
- (2012) Jordi Barretina et al. NATURE
- Differential Pathogenesis of Lung Adenocarcinoma Subtypes Involving Sequence Mutations, Copy Number, Chromosomal Instability, and Methylation
- (2012) Matthew D. Wilkerson et al. PLoS One
- Selective Killing of Mixed Lineage Leukemia Cells by a Potent Small-Molecule DOT1L Inhibitor
- (2011) Scott R. Daigle et al. CANCER CELL
- BET Bromodomain Inhibition as a Therapeutic Strategy to Target c-Myc
- (2011) Jake E. Delmore et al. CELL
- DNA Methylation and SETDB1/H3K9me3 Regulate Predominantly Distinct Sets of Genes, Retroelements, and Chimeric Transcripts in mESCs
- (2011) Mohammad M. Karimi et al. Cell Stem Cell
- Dual Functions of Histone-LysineN-Methyltransferase Setdb1 Protein at Promyelocytic Leukemia-Nuclear Body (PML-NB)
- (2011) Sunwha Cho et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- RNAi screen identifies Brd4 as a therapeutic target in acute myeloid leukaemia
- (2011) Johannes Zuber et al. NATURE
- Inhibition of BET recruitment to chromatin as an effective treatment for MLL-fusion leukaemia
- (2011) Mark A. Dawson et al. NATURE
- The histone methyltransferase SETDB1 is recurrently amplified in melanoma and accelerates its onset
- (2011) Craig J. Ceol et al. NATURE
- Genome-wide association study identifies a new melanoma susceptibility locus at 1q21.3
- (2011) Stuart MacGregor et al. NATURE GENETICS
- Cancer epigenetics reaches mainstream oncology
- (2011) Manuel Rodríguez-Paredes et al. NATURE MEDICINE
- Treatment of HER2-positive breast cancer: current status and future perspectives
- (2011) Carlos L. Arteaga et al. Nature Reviews Clinical Oncology
- Histone onco-modifications
- (2011) J Füllgrabe et al. ONCOGENE
- Silencing of Kruppel-like factor 2 by the histone methyltransferase EZH2 in human cancer
- (2011) H Taniguchi et al. ONCOGENE
- Somatic mutations at EZH2 Y641 act dominantly through a mechanism of selectively altered PRC2 catalytic activity, to increase H3K27 trimethylation
- (2010) D. B. Yap et al. BLOOD
- Aberrant Epigenetic Landscape in Cancer: How Cellular Identity Goes Awry
- (2010) María Berdasco et al. DEVELOPMENTAL CELL
- Key roles of histone methyltransferase and demethylase in leukemogenesis
- (2010) Akihide Yoshimi et al. JOURNAL OF CELLULAR BIOCHEMISTRY
- Proviral silencing in embryonic stem cells requires the histone methyltransferase ESET
- (2010) Toshiyuki Matsui et al. NATURE
- SetDB1 contributes to repression of genes encoding developmental regulators and maintenance of ES cell state
- (2009) S. Bilodeau et al. GENES & DEVELOPMENT
- Epigenetic inactivation of the Sotos overgrowth syndrome gene histone methyltransferase NSD1 in human neuroblastoma and glioma
- (2009) M. Berdasco et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- DNER, an Epigenetically Modulated Gene, Regulates Glioblastoma-Derived Neurosphere Cell Differentiation and Tumor Propagation
- (2009) Peng Sun et al. STEM CELLS
- Deregulation of histone lysine methyltransferases contributes to oncogenic transformation of human bronchoepithelial cells
- (2008) Hideo Watanabe et al. Cancer Cell International
- Oncogenic BRAF Induces Senescence and Apoptosis through Pathways Mediated by the Secreted Protein IGFBP7
- (2008) Narendra Wajapeyee et al. CELL
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