Prolactin suppresses a progestin-induced CK5-positive cell population in luminal breast cancer through inhibition of progestin-driven BCL6 expression
出版年份 2013 全文链接
标题
Prolactin suppresses a progestin-induced CK5-positive cell population in luminal breast cancer through inhibition of progestin-driven BCL6 expression
作者
关键词
-
出版物
ONCOGENE
Volume 33, Issue 17, Pages 2215-2224
出版商
Springer Nature
发表日期
2013-05-27
DOI
10.1038/onc.2013.172
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Transient Low Doses of DNA-Demethylating Agents Exert Durable Antitumor Effects on Hematological and Epithelial Tumor Cells
- (2012) Hsing-Chen Tsai et al. CANCER CELL
- Loss of Nuclear Localized and Tyrosine Phosphorylated Stat5 in Breast Cancer Predicts Poor Clinical Outcome and Increased Risk of Antiestrogen Therapy Failure
- (2011) Amy R. Peck et al. JOURNAL OF CLINICAL ONCOLOGY
- BCL6-mediated repression of p53 is critical for leukemia stem cell survival in chronic myeloid leukemia
- (2011) Christian Hurtz et al. JOURNAL OF EXPERIMENTAL MEDICINE
- Interplay between progesterone and prolactin in mammary development and implications for breast cancer
- (2011) Heather J. Lee et al. MOLECULAR AND CELLULAR ENDOCRINOLOGY
- The Induction of KLF5 Transcription Factor by Progesterone Contributes to Progesterone-Induced Breast Cancer Cell Proliferation and Dedifferentiation
- (2011) Rong Liu et al. MOLECULAR ENDOCRINOLOGY
- BCL6 enables Ph+ acute lymphoblastic leukaemia cells to survive BCR–ABL1 kinase inhibition
- (2011) Cihangir Duy et al. NATURE
- Maintenance of hormone responsiveness in luminal breast cancers by suppression of Notch
- (2011) J. M. Haughian et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- PTP1B Suppresses Prolactin Activation of Stat5 in Breast Cancer Cells
- (2010) Kevin J. Johnson et al. AMERICAN JOURNAL OF PATHOLOGY
- Cytokeratin 5 positive cells represent a steroid receptor negative and therapy resistant subpopulation in luminal breast cancers
- (2010) Peter Kabos et al. BREAST CANCER RESEARCH AND TREATMENT
- A Small-Molecule Inhibitor of BCL6 Kills DLBCL Cells In Vitro and In Vivo
- (2010) Leandro C. Cerchietti et al. CANCER CELL
- Prolactin Inhibits BCL6 Expression in Breast Cancer through a Stat5a-Dependent Mechanism
- (2010) T. H. Tran et al. CANCER RESEARCH
- Control of mammary stem cell function by steroid hormone signalling
- (2010) Marie-Liesse Asselin-Labat et al. NATURE
- Progesterone induces adult mammary stem cell expansion
- (2010) Purna A. Joshi et al. NATURE
- Reciprocal Effects of STAT5 and STAT3 in Breast Cancer
- (2009) S. R. Walker et al. MOLECULAR CANCER RESEARCH
- Protein kinases mediate ligand-independent derepression of sumoylated progesterone receptors in breast cancer cells
- (2009) A. R. Daniel et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A peptomimetic inhibitor of BCL6 with potent antilymphoma effects in vitro and in vivo
- (2008) L. C. Cerchietti et al. BLOOD
- Co-overexpression of Janus kinase 2 and signal transducer and activator of transcription 5a promotes differentiation of mammary cancer cells through reversal of epithelial–mesenchymal transition
- (2008) Ahmed S. Sultan et al. CANCER SCIENCE
- Transcription Factor Signal Transducer and Activator of Transcription 5 Promotes Growth of Human Prostate Cancer Cells In vivo
- (2008) A. Dagvadorj et al. CLINICAL CANCER RESEARCH
- Progestins in Hormone Replacement Therapies Reactivate Cancer Stem Cells in Women with Preexisting Breast Cancers: A Hypothesis
- (2008) Kathryn B. Horwitz et al. JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM
- Intrinsic Resistance of Tumorigenic Breast Cancer Cells to Chemotherapy
- (2008) X. Li et al. JNCI-Journal of the National Cancer Institute
- Rare steroid receptor-negative basal-like tumorigenic cells in luminal subtype human breast cancer xenografts
- (2008) K. B. Horwitz et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Discover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversationAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started