标题
ARAF acts as a scaffold to stabilize BRAF:CRAF heterodimers
作者
关键词
-
出版物
ONCOGENE
Volume 32, Issue 26, Pages 3207-3212
出版商
Springer Nature
发表日期
2012-08-27
DOI
10.1038/onc.2012.330
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Nilotinib and MEK Inhibitors Induce Synthetic Lethality through Paradoxical Activation of RAF in Drug-Resistant Chronic Myeloid Leukemia
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- c-Raf, but Not B-Raf, Is Essential for Development of K-Ras Oncogene-Driven Non-Small Cell Lung Carcinoma
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- RAF Inhibitor-Induced KSR1/B-RAF Binding and Its Effects on ERK Cascade Signaling
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- Improved Survival with Vemurafenib in Melanoma with BRAF V600E Mutation
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- Mutation that blocks ATP binding creates a pseudokinase stabilizing the scaffolding function of kinase suppressor of Ras, CRAF and BRAF
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- Acquired Resistance to BRAF Inhibitors Mediated by a RAF Kinase Switch in Melanoma Can Be Overcome by Cotargeting MEK and IGF-1R/PI3K
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- Kinase-Dead BRAF and Oncogenic RAS Cooperate to Drive Tumor Progression through CRAF
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- RAF inhibitors transactivate RAF dimers and ERK signalling in cells with wild-type BRAF
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- RAF inhibitors prime wild-type RAF to activate the MAPK pathway and enhance growth
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- Clinical efficacy of a RAF inhibitor needs broad target blockade in BRAF-mutant melanoma
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- Inhibition of Mutated, Activated BRAF in Metastatic Melanoma
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- Insights into the molecular function of the inactivating mutations of B-Raf involving the DFG motif
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- Impact of Feedback Phosphorylation and Raf Heterodimerization on Normal and Mutant B-Raf Signaling
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