Upregulation of VEGF-A and CD24 gene expression by the tGLI1 transcription factor contributes to the aggressive behavior of breast cancer cells
出版年份 2011 全文链接
标题
Upregulation of VEGF-A and CD24 gene expression by the tGLI1 transcription factor contributes to the aggressive behavior of breast cancer cells
作者
关键词
-
出版物
ONCOGENE
Volume 31, Issue 1, Pages 104-115
出版商
Springer Nature
发表日期
2011-06-13
DOI
10.1038/onc.2011.219
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Downregulation of Focal Adhesion Kinase (FAK) by cord blood stem cells inhibits angiogenesis in glioblastoma
- (2016) Venkata Ramesh Dasari et al. Aging-US
- EGFR and EGFRvIII undergo stress- and EGFR kinase inhibitor-induced mitochondrial translocalization: A potential mechanism of EGFR-driven antagonism of apoptosis
- (2011) Xinyu Cao et al. Molecular Cancer
- EGFR and EGFRvIII interact with PUMA to inhibit mitochondrial translocalization of PUMA and PUMA-mediated apoptosis independent of EGFR kinase activity
- (2010) Hu Zhu et al. CANCER LETTERS
- CD44posCD49fhiCD133/2hi Defines Xenograft-Initiating Cells in Estrogen Receptor–Negative Breast Cancer
- (2010) Matthew J. Meyer et al. CANCER RESEARCH
- The Human Glioma-Associated Oncogene Homolog 1 (GLI1) Family of Transcription Factors in Gene Regulation and Diseases
- (2010) Hu Zhu et al. CURRENT GENOMICS
- Targeting Breast Cancer Stem Cells
- (2010) Suling Liu et al. JOURNAL OF CLINICAL ONCOLOGY
- Cyclooxygenase-2 Is a Novel Transcriptional Target of the Nuclear EGFR-STAT3 and EGFRvIII-STAT3 Signaling Axes
- (2010) H.-W. Lo et al. MOLECULAR CANCER RESEARCH
- More than Markers: Biological Significance of Cancer Stem Cell-Defining Molecules
- (2010) S. B. Keysar et al. MOLECULAR CANCER THERAPEUTICS
- Breast cancer stem cells: tools and models to rely on
- (2009) Emmanuelle Charafe-Jauffret et al. BMC CANCER
- Expression of the glioma-associated oncogene homolog (GLI) 1in human breast cancer is associated with unfavourable overall survival
- (2009) Anette ten Haaf et al. BMC CANCER
- RETRACTED ARTICLE: FoxM1 down-regulation leads to inhibition of proliferation, migration and invasion of breast cancer cells through the modulation of extra-cellular matrix degrading factors
- (2009) Aamir Ahmad et al. BREAST CANCER RESEARCH AND TREATMENT
- Development of Mammary Tumors by Conditional Expression of GLI1
- (2009) M. Fiaschi et al. CANCER RESEARCH
- A Novel Splice Variant of GLI1 That Promotes Glioblastoma Cell Migration and Invasion
- (2009) H.-W. Lo et al. CANCER RESEARCH
- Hedgehog signalling in breast cancer
- (2009) M. Kasper et al. CARCINOGENESIS
- Tumor metabolism of lactate: the influence and therapeutic potential for MCT and CD147 regulation
- (2009) Kelly M Kennedy et al. Future Oncology
- Activation of the hedgehog-signaling pathway in human cancer and the clinical implications
- (2009) L Yang et al. ONCOGENE
- Molecular Profiling of Breast Cancer Cell Lines Defines Relevant Tumor Models and Provides a Resource for Cancer Gene Discovery
- (2009) Jessica Kao et al. PLoS One
- Hedgehog signalling in vascular development
- (2008) Takashi Nagase et al. ANGIOGENESIS
- Constitutively Activated STAT3 Frequently Coexpresses with Epidermal Growth Factor Receptor in High-Grade Gliomas and Targeting STAT3 Sensitizes Them to Iressa and Alkylators
- (2008) H.-W. Lo et al. CLINICAL CANCER RESEARCH
- Novel Human Glioma-associated Oncogene 1 (GLI1) Splice Variants Reveal Distinct Mechanisms in the Terminal Transduction of the Hedgehog Signal
- (2008) Takashi Shimokawa et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Identification and Functional Characterization of the Human Glutathione S-Transferase P1 Gene as a Novel Transcriptional Target of the p53 Tumor Suppressor Gene
- (2008) H.-W. Lo et al. MOLECULAR CANCER RESEARCH
- CD24 induces localization of β1 integrin to lipid raft domains
- (2007) Steffen Runz et al. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreDiscover Peeref hubs
Discuss science. Find collaborators. Network.
Join a conversation