Pbx1 is a downstream target of Evi-1 in hematopoietic stem/progenitors and leukemic cells

EVI1 Impairs Myelopoiesis by Deregulation of PU.1 Function

(2009) Leopoldo Laricchia-Robbio et al.   CANCER RESEARCH

Pathogenetic significance of ecotropic viral integration site-1 in hematological malignancies

(2009) Susumu Goyama et al.   CANCER SCIENCE

High EVI1 levels predict adverse outcome in acute myeloid leukemia: prevalence of EVI1 overexpression and chromosome 3q26 abnormalities underestimated

(2008) S. Lugthart et al.   BLOOD

AML1 mutations induced MDS and MDS/AML in a mouse BMT model

(2008) N. Watanabe-Okochi et al.   BLOOD

Identification ofPbx1, a Potential Oncogene, as a Notch3 Target Gene in Ovarian Cancer

(2008) Joon T. Park et al.   CANCER RESEARCH

Evi-1 promotes para-aortic splanchnopleural hematopoiesis through up-regulation of GATA-2 and repression of TGF-b signaling

(2008) Tomohiko Sato et al.   CANCER SCIENCE

Evi-1 Is a Critical Regulator for Hematopoietic Stem Cells and Transformed Leukemic Cells

(2008) Susumu Goyama et al.   Cell Stem Cell

Pbx1 Regulates Self-Renewal of Long-Term Hematopoietic Stem Cells by Maintaining Their Quiescence

(2008) Francesca Ficara et al.   Cell Stem Cell

AML1-Evi-1 specifically transforms hematopoietic stem cells through fusion of the entire Evi-1 sequence to AML1

(2008) M Takeshita et al.   LEUKEMIA

Significant increase of self-renewal in hematopoietic cells after forced expression of EVI1

(2007) Leopoldo Laricchia-Robbio et al.   BLOOD CELLS MOLECULES AND DISEASES