Upregulation of BACE1 and β-Amyloid Protein Mediated by Chronic Cerebral Hypoperfusion Contributes to Cognitive Impairment and Pathogenesis of Alzheimer’s Disease

标题
Upregulation of BACE1 and β-Amyloid Protein Mediated by Chronic Cerebral Hypoperfusion Contributes to Cognitive Impairment and Pathogenesis of Alzheimer’s Disease
作者
关键词
-
出版物
NEUROCHEMICAL RESEARCH
Volume 34, Issue 7, Pages 1226-1235
出版商
Springer Nature
发表日期
2009-01-06
DOI
10.1007/s11064-008-9899-y

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