Mechanisms for U2AF to define 3′ splice sites and regulate alternative splicing in the human genome
出版年份 2014 全文链接
标题
Mechanisms for U2AF to define 3′ splice sites and regulate alternative splicing in the human genome
作者
关键词
-
出版物
NATURE STRUCTURAL & MOLECULAR BIOLOGY
Volume 21, Issue 11, Pages 997-1005
出版商
Springer Nature
发表日期
2014-10-20
DOI
10.1038/nsmb.2906
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Context-dependent control of alternative splicing by RNA-binding proteins
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- Nuclear Matrix Factor hnRNP U/SAF-A Exerts a Global Control of Alternative Splicing by Regulating U2 snRNP Maturation
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- The Akt-SRPK-SR Axis Constitutes a Major Pathway in Transducing EGF Signaling to Regulate Alternative Splicing in the Nucleus
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- Using positional distribution to identify splicing elements and predict pre-mRNA processing defects in human genes
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- The U2AF35-related protein Urp contacts the 3' splice site to promote U12-type intron splicing and the second step of U2-type intron splicing
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- Genomic mRNA Profiling Reveals Compensatory Mechanisms for the Requirement of the Essential Splicing Factor U2AF
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