期刊
NATURE MEDICINE
卷 18, 期 6, 页码 858-859出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/nm.2797
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资金
- NINDS NIH HHS [R01 NS034179] Funding Source: Medline
How blood-borne inflammatory cells cause tissue damage in the brain after ischemic stroke remains elusive. Peroxiredoxins, cytosolic antioxidant proteins vital for redox balance, are released extracellularly from ischemic cells, acting as potent 'danger signals' that activate macrophages and lead to a harmful cytokine response, a new study shows. The findings unveil a new culprit in the delayed phase of ischemic injury and suggest new therapeutic approaches (pages 911-917).
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