The psychiatric disease risk factors DISC1 and TNIK interact to regulate synapse composition and function
出版年份 2010 全文链接
标题
The psychiatric disease risk factors DISC1 and TNIK interact to regulate synapse composition and function
作者
关键词
-
出版物
MOLECULAR PSYCHIATRY
Volume 16, Issue 10, Pages 1006-1023
出版商
Springer Nature
发表日期
2010-09-14
DOI
10.1038/mp.2010.87
参考文献
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注意:仅列出部分参考文献,下载原文获取全部文献信息。- Disrupted-in-Schizophrenia 1 (DISC1) regulates spines of the glutamate synapse via Rac1
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- Structural dynamics of dendritic spines in memory and cognition
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- Disrupted in Schizophrenia 1 Regulates Neuronal Progenitor Proliferation via Modulation of GSK3β/β-Catenin Signaling
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- The kinase TNIK is an essential activator of Wnt target genes
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- DISC1, PDE4B, and NDE1 at the centrosome and synapse
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- MINK is a Rap2 effector for phosphorylation of the postsynaptic scaffold protein TANC1
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- Ndel1 alters its conformation by sequestering cAMP-specific phosphodiesterase-4D3 (PDE4D3) in a manner that is dynamically regulated through Protein Kinase A (PKA)
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- Role of glutamate in schizophrenia: integrating excitatory avenues of research
- (2008) Monsheel Sodhi et al. Expert Review of Neurotherapeutics
- Targeting the glutamatergic system to develop novel, improved therapeutics for mood disorders
- (2008) Gerard Sanacora et al. NATURE REVIEWS DRUG DISCOVERY
- Actin in action: the interplay between the actin cytoskeleton and synaptic efficacy
- (2008) Lorenzo A. Cingolani et al. NATURE REVIEWS NEUROSCIENCE
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- Dendritic spine dynamics – a key role for kalirin-7
- (2008) Peter Penzes et al. TRENDS IN NEUROSCIENCES
- How has DISC1 enabled drug discovery?
- (2007) Qi Wang et al. MOLECULAR AND CELLULAR NEUROSCIENCE
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