Amyloid peptides ABri and ADan show differential neurotoxicity in transgenic Drosophila models of familial British and Danish dementia
出版年份 2014 全文链接
标题
Amyloid peptides ABri and ADan show differential neurotoxicity in transgenic Drosophila models of familial British and Danish dementia
作者
关键词
Alzheimer’s disease, Familial British dementia, Familial Danish dementia, ABri, ADan, Neurotoxicity, <em class=EmphasisTypeItalic >Drosophila</em>
出版物
Molecular Neurodegeneration
Volume 9, Issue 1, Pages 5
出版商
Springer Nature
发表日期
2014-01-10
DOI
10.1186/1750-1326-9-5
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Increased Tau Phosphorylation and Tau Truncation, and Decreased Synaptophysin Levels in Mutant BRI2/Tau Transgenic Mice
- (2013) Holly J. Garringer et al. PLoS One
- Probing Mechanisms That Underlie Human Neurodegenerative Diseases inDrosophila
- (2012) M. Jaiswal et al. Annual Review of Genetics
- Ageing Increases Vulnerability to Aβ42 Toxicity in Drosophila
- (2012) Iain Rogers et al. PLoS One
- Genetic Inhibition of Solute-Linked Carrier 39 Family Transporter 1 Ameliorates Aβ Pathology in a Drosophila Model of Alzheimer's Disease
- (2012) Minglin Lang et al. PLoS Genetics
- The Central Clock Neurons Regulate Lipid Storage in Drosophila
- (2011) Justin R. DiAngelo et al. PLoS One
- Activation of JNK Signaling Mediates Amyloid-ß-Dependent Cell Death
- (2011) Meghana Tare et al. PLoS One
- Expression of -Amyloid Induced Age-Dependent Presynaptic and Axonal Changes in Drosophila
- (2010) X.-L. Zhao et al. JOURNAL OF NEUROSCIENCE
- Memory Deficits Due to Familial British Dementia BRI2 Mutation Are Caused by Loss of BRI2 Function Rather than Amyloidosis
- (2010) R. Tamayev et al. JOURNAL OF NEUROSCIENCE
- Modeling familial Danish dementia in mice supports the concept of the amyloid hypothesis of Alzheimer's disease
- (2010) Janaky Coomaraswamy et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- PI3 kinase signaling is involved in A -induced memory loss in Drosophila
- (2010) H.-C. Chiang et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Modeling familial British and Danish dementia
- (2009) Holly J. Garringer et al. Brain Structure & Function
- Generation and Initial Characterization of FDD Knock In Mice
- (2009) Luca Giliberto et al. PLoS One
- Mitochondrial Mislocalization Underlies Aβ42-Induced Neuronal Dysfunction in a Drosophila Model of Alzheimer's Disease
- (2009) Kanae Iijima-Ando et al. PLoS One
- DrosophilaModels of Neurodegenerative Diseases
- (2008) Bingwei Lu et al. Annual Review of Pathology-Mechanisms of Disease
- Cerebral Amyloid Angiopathy and Parenchymal Amyloid Deposition in Transgenic Mice Expressing the Danish Mutant Form of Human BRI2
- (2008) Ruben Vidal et al. BRAIN PATHOLOGY
- Identification of Novel Genes That Modify Phenotypes Induced by Alzheimer's -Amyloid Overexpression in Drosophila
- (2008) W. Cao et al. GENETICS
- Overexpression of Neprilysin Reduces Alzheimer Amyloid-β42 (Aβ42)-induced Neuron Loss and Intraneuronal Aβ42 Deposits but Causes a Reduction in cAMP-responsive Element-binding Protein-mediated Transcription, Age-dependent Axon Pathology, and Premature Death inDrosophila
- (2008) Kanae Iijima-Ando et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- BRI2 (ITM2b) Inhibits A Deposition In Vivo
- (2008) J. Kim et al. JOURNAL OF NEUROSCIENCE
- Aβ42 Mutants with Different Aggregation Profiles Induce Distinct Pathologies in Drosophila
- (2008) Koichi Iijima et al. PLoS One
- A Functional Misexpression Screen Uncovers a Role for Enabled in Progressive Neurodegeneration
- (2008) Carolina Rezával et al. PLoS One
- The Toll→NFκB Signaling Pathway Mediates the Neuropathological Effects of the Human Alzheimer's Aβ42 Polypeptide in Drosophila
- (2008) Lihua Tan et al. PLoS One
Find Funding. Review Successful Grants.
Explore over 25,000 new funding opportunities and over 6,000,000 successful grants.
ExploreBecome a Peeref-certified reviewer
The Peeref Institute provides free reviewer training that teaches the core competencies of the academic peer review process.
Get Started