Rac1 Activation in Podocytes Induces Rapid Foot Process Effacement and Proteinuria

ARHGDIA mutations cause nephrotic syndrome via defective RHO GTPase signaling

(2013) Heon Yung Gee et al.   JOURNAL OF CLINICAL INVESTIGATION

ARHGDIA: a novel gene implicated in nephrotic syndrome

(2013) Indra Rani Gupta et al.   JOURNAL OF MEDICAL GENETICS

Divergent functions of the Rho GTPases Rac1 and Cdc42 in podocyte injury

(2013) Simone M. Blattner et al.   KIDNEY INTERNATIONAL

Nanoscale protein architecture of the kidney glomerular basement membrane

(2013) Hani Suleiman et al.   eLife

Podocyte-Specific Loss of Cdc42 Leads to Congenital Nephropathy

(2012) R. P. Scott et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Mechanisms of the proteinuria induced by Rho GTPases

(2012) Liming Wang et al.   KIDNEY INTERNATIONAL

Cell Biology and Pathology of Podocytes

(2011) Anna Greka et al.   Annual Review of Physiology

Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis

(2011) Shreeram Akilesh et al.   JOURNAL OF CLINICAL INVESTIGATION

Activation of RhoA in Podocytes Induces Focal Segmental Glomerulosclerosis

(2011) L. Zhu et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Superresolution Imaging of Chemical Synapses in the Brain

(2010) Adish Dani et al.   NEURON

Parietal Epithelial Cells Regenerate Podocytes

(2009) R. Poulsom et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Proteinuria: an enzymatic disease of the podocyte?

(2009) Peter Mundel et al.   KIDNEY INTERNATIONAL

Recruitment of Podocytes from Glomerular Parietal Epithelial Cells

(2008) D. Appel et al.   JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY

Modification of mineralocorticoid receptor function by Rac1 GTPase: implication in proteinuric kidney disease

(2008) Shigeru Shibata et al.   NATURE MEDICINE