期刊
KIDNEY INTERNATIONAL
卷 78, 期 4, 页码 333-335出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/ki.2010.182
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T cells have a well-described role in renal injury, which leads to the secondary development of renal fibrosis. It has generally been assumed that this fibrotic response is an indirect consequence of T cell-mediated renal injury, rather than T cells' being directly involved in fibrosis. Tapmeier et al. now provide evidence that CD4(+) T cells promote interstitial fibrosis in the obstructed mouse kidney in a mechanism that appears to dissociate myofibroblast accumulation and matrix deposition. Kidney International (2010) 78, 333-335. doi:10.1038/ki.2010.182
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