期刊
KIDNEY INTERNATIONAL
卷 74, 期 3, 页码 257-258出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/ki.2008.243
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资金
- NIDDK NIH HHS [R01 DK063114] Funding Source: Medline
Acute kidney injury following folate administration is characterized by a vascular remodeling that is initially proliferative but subsequently results in vascular endothelial loss. Interventions directed toward promoting endothelial growth may preserve vascular structure and therefore renal function. However, angiopoietin-1 therapy in the setting of folate-induced acute kidney injury resulted in an expanded fibrotic response despite apparent preservation of the vasculature, indicating that renal repair responses are complex and vascular-directed therapies should be approached with caution.
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