Expression of Neuronal CXCL10 Induced by Rabies Virus Infection Initiates Infiltration of Inflammatory Cells, Production of Chemokines and Cytokines, and Enhancement of Blood-Brain Barrier Permeability

Enhancement of Blood-Brain Barrier Permeability and Reduction of Tight Junction Protein Expression Are Modulated by Chemokines/Cytokines Induced by Rabies Virus Infection

(2014) Q. Chai et al.   JOURNAL OF VIROLOGY

K63-linked polyubiquitination of transcription factor IRF1 is essential for IL-1-induced production of chemokines CXCL10 and CCL5

(2014) Kuzhuvelil B Harikumar et al.   NATURE IMMUNOLOGY

Review: The chemokine receptor CXCR3 and its ligands CXCL9, CXCL10 and CXCL11 in neuroimmunity - a tale of conflict and conundrum

(2010) M. Müller et al.   NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY

Cellular mechanisms of IL-17-induced blood-brain barrier disruption

(2009) Jula Huppert et al.   FASEB JOURNAL

Impact of Tumor Angiogenesis in Peritoneal Mesothelioma After Radical Cytoreduction and Hyperthermic Intraperitoneal Chemotherapy

(2009) Terence C. Chua et al.   PATHOLOGY & ONCOLOGY RESEARCH

Role of chemokines in the enhancement of BBB permeability and inflammatory infiltration after rabies virus infection

(2009) Yi Kuang et al.   VIRUS RESEARCH

The Production of Antibody by Invading B Cells Is Required for the Clearance of Rabies Virus from the Central Nervous System

(2009) D. Craig Hooper et al.   PLoS Neglected Tropical Diseases

CXCL10-Producing Mucosal CD4+ T Cells, NK Cells, and NKT Cells Are Associated with Chronic Colitis in IL-10−/− Mice, Which Can Be Abrogated by Anti-CXCL10 Antibody Inhibition

(2008) Udai P. Singh et al.   JOURNAL OF INTERFERON AND CYTOKINE RESEARCH

Interleukin-17 Production in Central Nervous System-Infiltrating T Cells and Glial Cells Is Associated with Active Disease in Multiple Sclerosis

(2007) John S. Tzartos et al.   AMERICAN JOURNAL OF PATHOLOGY