标题
Circadian control of innate immunity in macrophages by miR-155 targetingBmal1
作者
关键词
-
出版物
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
Volume 112, Issue 23, Pages 7231-7236
出版商
Proceedings of the National Academy of Sciences
发表日期
2015-05-21
DOI
10.1073/pnas.1501327112
参考文献
相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。- Circadian Clock Proteins and Immunity
- (2014) Anne M. Curtis et al. IMMUNITY
- Identification of an NF-κB p50/p65-responsive site in the human MIR155HG promoter
- (2013) Ryan C Thompson et al. BMC MOLECULAR BIOLOGY
- The Role of Ets2 Transcription Factor in the Induction of MicroRNA-155 (miR-155) by Lipopolysaccharide and Its Targeting by Interleukin-10
- (2013) Susan R. Quinn et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription
- (2013) Michael T. Y. Lam et al. NATURE
- Circadian clock regulates the host response to Salmonella
- (2013) M. M. Bellet et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Circadian Gene Bmal1 Regulates Diurnal Oscillations of Ly6Chi Inflammatory Monocytes
- (2013) K. D. Nguyen et al. SCIENCE
- Knitting Up the Raveled Sleave of Care
- (2013) G. Yang et al. Science Translational Medicine
- Clock-controlled mir-142-3p can target its activator, Bmal1
- (2012) Xiaochao Tan et al. BMC MOLECULAR BIOLOGY
- The Circadian Clock Controls Toll-like Receptor 9-Mediated Innate and Adaptive Immunity
- (2012) Adam C. Silver et al. IMMUNITY
- Adrenergic Nerves Govern Circadian Leukocyte Recruitment to Tissues
- (2012) Christoph Scheiermann et al. IMMUNITY
- MicroRNA-155 promotes atherosclerosis by repressing Bcl6 in macrophages
- (2012) Maliheh Nazari-Jahantigh et al. JOURNAL OF CLINICAL INVESTIGATION
- Micro-Managing the Circadian Clock: The Role of microRNAs in Biological Timekeeping
- (2012) Neel Mehta et al. JOURNAL OF MOLECULAR BIOLOGY
- Core circadian protein CLOCK is a positive regulator of NF- B-mediated transcription
- (2012) M. L. Spengler et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- miR-221 and miR-155 regulate human dendritic cell development, apoptosis, and IL-12 production through targeting of p27kip1, KPC1, and SOCS-1
- (2011) C. Lu et al. BLOOD
- The nuclear receptor REV-ERB mediates circadian regulation of innate immunity through selective regulation of inflammatory cytokines
- (2011) J. E. Gibbs et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Up-regulation of MicroRNA-155 in Macrophages Contributes to Increased Tumor Necrosis Factor α (TNFα) Production via Increased mRNA Half-life in Alcoholic Liver Disease
- (2010) Shashi Bala et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- IL-10 Inhibits miR-155 Induction by Toll-like Receptors
- (2010) Claire E. McCoy et al. JOURNAL OF BIOLOGICAL CHEMISTRY
- Dysregulation of Inflammatory Responses by Chronic Circadian Disruption
- (2010) O. Castanon-Cervantes et al. JOURNAL OF IMMUNOLOGY
- Integration of microRNA miR-122 in hepatic circadian gene expression
- (2009) D. Gatfield et al. GENES & DEVELOPMENT
- A role for microRNAs in the Drosophila circadian clock
- (2009) S. Kadener et al. GENES & DEVELOPMENT
- Inositol phosphatase SHIP1 is a primary target of miR-155
- (2009) R. M. O'Connell et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- A circadian clock in macrophages controls inflammatory immune responses
- (2009) Maren Keller et al. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA
- Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?
- (2008) Witold Filipowicz et al. NATURE REVIEWS GENETICS
Publish scientific posters with Peeref
Peeref publishes scientific posters from all research disciplines. Our Diamond Open Access policy means free access to content and no publication fees for authors.
Learn MoreAsk a Question. Answer a Question.
Quickly pose questions to the entire community. Debate answers and get clarity on the most important issues facing researchers.
Get Started