SATB1 is overexpressed in metastatic prostate cancer and promotes prostate cancer cell growth and invasion

Upregulation of SATB1 Is Associated with Prostate Cancer Aggressiveness and Disease Progression

(2013) Sanjeev Shukla et al.   PLoS One

The current role of circulating tumor cells in the diagnosis and management of bone metastases in advanced prostate cancer

(2012) Fred Saad et al.   Future Oncology

A novel patient-derived intra-femoral xenograft model of bone metastatic prostate cancer that recapitulates mixed osteolytic and osteoblastic lesions

(2012) Omer Raheem et al.   Journal of Translational Medicine

Upregulation of SATB1 is associated with the development and progression of glioma

(2012) Sheng-Hua Chu et al.   Journal of Translational Medicine

The Role of Nuclear Matrix Proteins Binding to Matrix Attachment Regions (MARs) in Prostate Cancer Cell Differentiation

(2012) Paola Barboro et al.   PLoS One

Correlation of SATB1 overexpression with the progression of human rectal cancer

(2011) Wen-Jian Meng et al.   INTERNATIONAL JOURNAL OF COLORECTAL DISEASE

N-terminal PDZ-like domain of chromatin organizer SATB1 contributes towards its function as transcription regulator

(2011) Dimple Notani et al.   JOURNAL OF BIOSCIENCES

Clinicopathologic and prognostic significance of SATB1 in cutaneous malignant melanoma

(2011) Hongxiang Chen et al.   JOURNAL OF DERMATOLOGICAL SCIENCE

The BCL2 gene is regulated by a special AT-rich sequence binding protein 1-mediated long range chromosomal interaction between the promoter and the distal element located within the 3′-UTR

(2011) Feiran Gong et al.   NUCLEIC ACIDS RESEARCH

Involvement of NF-κB/miR-448 regulatory feedback loop in chemotherapy-induced epithelial–mesenchymal transition of breast cancer cells

(2010) Q-Q Li et al.   CELL DEATH AND DIFFERENTIATION

Acetylation-Dependent Interaction of SATB1 and CtBP1 Mediates Transcriptional Repression by SATB1

(2008) P. K. Purbey et al.   MOLECULAR AND CELLULAR BIOLOGY

SATB1 reprogrammes gene expression to promote breast tumour growth and metastasis

(2008) Hye-Jung Han et al.   NATURE